An Emergency Medicine Broadsheet
·Phoenix·
Est. MMXXVI
Blue Fish Med · Today's Topic
Nausea, Vomiting and Hyperemesis in Pregnancy
Persistent vomiting in pregnancy can quietly become dehydration, electrolyte derangement, malnutrition, and Wernicke encephalopathy. Missing an alternate diagnosis — or under-treating hyperemesis — harms both parent and fetus.
A 27-year-old at 10 weeks’ gestation sits curled on the stretcher, lips cracked, ginger ale untouched at her feet. Every few minutes she dry-heaves into an emesis bag, then stares at the wall, dizzy when she tries to sit up. Her partner says she has not kept down food for days and the smell of coffee made her run to the bathroom. She is asking for “something safe for the baby,” but first you need to decide whether this is routine morning sickness or something much more dangerous.
— What’s your move? Read on.
Before you read
What dangerous mimics must be excluded before labeling this “just vomiting”?
Which antiemetics are reasonable in pregnancy, and when do you add thiamine, fluids, or admission?
When to Think of It
Think of this when a pregnant patient, especially in the first trimester, has persistent nausea/vomiting with inability to tolerate PO, weight loss, ketonuria, dehydration, orthostasis, or repeated ED visits. Escalate the concern if there is abdominal pain, fever, vaginal bleeding, focal peritoneal findings, or neurologic symptoms.
Sick or Not Sick
The key fork is well-appearing and tolerating fluids vs. dehydrated/ketotic/failed PO with electrolyte abnormalities or inability to protect nutrition. If she cannot keep down liquids, has significant dehydration, or you suspect another abdominal or obstetric process, she is not a discharge-from-triage patient.
The First Fifteen Minutes
Check vitals, orthostatics, bedside glucose, urine ketones, and fetal/obstetric status if appropriate gestational age/symptoms.
Normal saline 1–2 L IV bolus for dehydration/orthostasis, because volume repletion improves perfusion and reduces ketogenesis.
Dextrose-containing fluids only after thiamine if prolonged poor intake or concern for starvation; e.g., thiamine 100 mg IV now before dextrose, because it prevents precipitating Wernicke encephalopathy.
Pyridoxine (vitamin B6) 10–25 mg PO/IV every 8 hours for nausea, because it is safe and may reduce symptoms.
Doxylamine 12.5 mg PO every 6–8 hours or doxylamine-pyridoxine delayed release 10/10 mg PO (per product directions), because antihistamine antiemetic effect helps mild-to-moderate nausea.
Metoclopramide 10 mg IV/PO every 6 hours PRN for ongoing emesis, because dopamine antagonism improves gastric emptying and nausea.
Ondansetron 4–8 mg IV/PO every 6–8 hours PRN if symptoms persist, because 5-HT3 blockade is effective for refractory vomiting.
Prochlorperazine 5–10 mg IV/IM/PO every 6–8 hours PRN or promethazine 12.5–25 mg IV/IM/PO every 4–6 hours PRN if needed, because dopamine/antihistamine effects can control severe symptoms; monitor for sedation and dystonia.
Acetaminophen 650–1000 mg PO/IV if headache or discomfort is contributing, because pain worsens nausea; avoid NSAIDs in later pregnancy unless clearly indicated.
Definitive Care & Disposition
Treat reversible triggers: urinary infection, gastroenteritis, DKA, hepatitis, pancreatitis, appendicitis, ovarian pathology, or pregnancy complications. If she cannot tolerate PO after ED therapy, has electrolyte abnormalities, ketonuria with ongoing vomiting, weight loss, or recurrent visits, admit or place in observation for serial antiemetics, IV fluids, electrolyte correction, and nutrition support; severe refractory cases may need enteral feeding. OB involvement is appropriate for persistent hyperemesis, diagnostic uncertainty, or any concern for maternal-fetal compromise.
How This One Kills
The classic failure is anchoring on “morning sickness” and missing ectopic pregnancy, DKA, bowel obstruction, appendicitis, pyelonephritis, or cannabis hyperemesis — or giving dextrose before thiamine in a starved patient and triggering Wernicke injury.
The Differential — What Else Looks Like This
Cannabis Hyperemesis Syndrome — cyclic vomiting with chronic cannabis use, hot shower relief, and poor response to standard antiemetics; confusing it delays cessation counseling and symptom control.
Ectopic Pregnancy — pregnancy + abdominal pain, bleeding, syncope, or shoulder pain; missing it risks rupture and hemorrhagic shock.
Appendicitis — localized RLQ pain, fever, peritoneal signs; mislabeling it as hyperemesis delays surgery and increases perforation risk.
Pyelonephritis — fever, flank pain, urinary symptoms, CVA tenderness; confusing it with pregnancy vomiting misses sepsis and need for antibiotics.
The Second-Day Story
Hyperemesis does not always arrive as dramatic nonstop vomiting. Some patients are older, already partially treated, or have learned to sip just enough to avoid collapse, so the story becomes “can’t eat,” “everything tastes wrong,” or “I feel weak and dizzy when I stand.” Others present late with constipation, tachycardia, scant urine, or weight loss more than active emesis. The diagnosis is still clinical, but the warning lights are dehydration, ketones, and failure to function, not the volume of vomit on the floor.
Back to Our Patient
Back to our patient: the 27-year-old at 10 weeks with cracked lips, dry heaving, and dizziness is most consistent with hyperemesis gravidarum after you confirm no abdominal tenderness, fever, vaginal bleeding, or other red flags. She is risk-stratified as not safe for a quick discharge because she cannot tolerate PO and appears dehydrated, so you start IV fluids, give thiamine 100 mg IV before any dextrose, and add an antiemetic such as metoclopramide or ondansetron. If she improves, can drink, and labs are reassuring, she can go home with scheduled antiemetics and OB follow-up; if not, she needs observation or admission for ongoing IV therapy and electrolyte correction.
Patient Presentation to Attending
How you’d present this patient on the floor — tight, pertinent positives and negatives, no rambling
27-year-old G1P0 at 10 weeks by LMP with 4 days of persistent nausea and vomiting, unable to keep down food or liquids, here for dizziness and dehydration. She denies abdominal pain, vaginal bleeding, fever, diarrhea, dysuria, or focal neurologic symptoms, and she says this is worse than her baseline morning nausea. On exam she is tachycardic, dry-appearing, with benign abdomen and no peritoneal signs; urine shows ketones and her glucose is normal. I’m concerned for hyperemesis gravidarum versus a pregnancy-related or abdominal mimic, and I’d like IV fluids, thiamine before dextrose, antiemetics, electrolyte evaluation, and OB follow-up or observation if she can’t tolerate PO.
Study Directive
Memorize a stepwise ED algorithm for vomiting in pregnancy: confirm gestational age, screen for red flags, check hydration/ketones/glucose, treat with fluids + antiemetics, then decide discharge vs admit.
Write from memory the first-line pregnancy antiemetic options and their standard doses.
Nelson-Piercy C, Dean C, Shehmar M, et al. · BJOG, 2024 · PMID 38311315 · cited 117×
Authoritative pregnancy-specific guideline for ED management: risk-stratify NVP versus hyperemesis, check dehydration/electrolytes/ketones, give thiamine before dextrose, use stepwise safe antiemetics, and admit when oral intake or metaboli
PUL is not a diagnosis you can ignore; it is a temporary label that can hide ectopic pregnancy. The danger is premature reassurance in a patient whose...
A 31-year-old with a faint positive home test sits on the edge of the bed, one hand on her lower abdomen, the other clutching a paper towel that has a single pink spot on it. The ultrasound screen behind you is gray and indecisive: no clear intrauterine pregnancy, no obvious free fluid, and nothing dramatic enough to relax. She looks stable, but her pulse keeps climbing a little every time the word “where” comes up. You need to answer the question that matters most before she walks out the door.
Before You Read
What exactly counts as a pregnancy of unknown location?
Who can be discharged, and who needs serial testing or immediate OB/surgical escalation?
When does “unknown location” become “ectopic until proven otherwise”?
Why It Matters
PUL is not a diagnosis you can ignore; it is a temporary label that can hide ectopic pregnancy. The danger is premature reassurance in a patient whose pregnancy location has not yet been proven.
When to Think of It
Use this term when pregnancy test is positive but transvaginal ultrasound does not show a definitive intrauterine or ectopic pregnancy. It often presents with pain, spotting, or early pregnancy symptoms, but may also be minimally symptomatic.
Sick or Not Sick
The key fork is hemodynamically unstable or peritoneal vs. stable. Unstable patients are ectopic until proven otherwise and need immediate OB/surgical action; stable patients need quantitative hCG, ultrasound correlation, and reliable follow-up.
The First Fifteen Minutes
Assess hemodynamics immediately; establish IV access and type & screen/CBC.
Normal saline or Lactated Ringer’s 1–2 L IV bolus if tachycardic or volume depleted, because perfusion buys time while you clarify location.
Rh(D) immune globulin 50 mcg IM if Rh-negative and first-trimester bleeding/ectopic concern (use 300 mcg IM if smaller dose unavailable or later gestation), because it prevents alloimmunization.
Acetaminophen 650–1000 mg PO/IV for pain if mild, because it avoids masking deterioration better than sedating agents.
Avoid empiric methotrexate, and avoid “false reassurance” discharge until follow-up is secured; the next step is serial hCG and repeat TVUS, not a one-shot answer.
If unstable with concern for rupture: activate OB/GYN and surgery immediately; prepare blood products. No medication substitutes for source control in rupture.
Definitive Care & Disposition
Stable PUL requires serial quantitative hCG in ~48 hours plus repeat transvaginal ultrasound based on hCG trajectory and symptoms, with strict return precautions. A rising hCG that fails to appropriately declare an intrauterine pregnancy keeps ectopic on the table; falling hCG may indicate early loss but still needs follow-up to zero. Admit or observe patients with significant pain, poor follow-up, concerning hCG trends, or diagnostic uncertainty. If ectopic is confirmed or rupture is suspected, OB/GYN determines medical vs operative management, but instability or hemoperitoneum goes to the OR.
How This One Kills
The lethal error is calling an indeterminate scan “just early pregnancy” and losing the ectopic until it ruptures at home. The other common miss is anchoring on hCG alone without clinical status; a stable number does not rescue an unstable patient.
The Atypical Presentation
Some patients with PUL look deceptively well: mild spotting, vague cramping, a reassuring exam, and only a slightly positive test. Others have pain out of proportion to exam, shoulder-tip discomfort, or presyncope before any ultrasound clue appears. The degraded-signal version is the patient who is stable now, has no free fluid, and still leaves with a “maybe early pregnancy” label; if you cannot prove an intrauterine pregnancy, you have not finished the job.
Back to Our Patient
Back to our patient: the 31-year-old with a faint positive test and spotting has a pregnancy of unknown location because the ultrasound does not yet prove an intrauterine pregnancy or ectopic. She is stable but not discharged blindly; you check Rh status, give RhIg if indicated, obtain quantitative hCG and baseline labs, and arrange 48-hour follow-up with repeat hCG and ultrasound instructions. If her pain worsens, she becomes dizzy, or she cannot ensure follow-up, she stays for observation and OB involvement.
Patient Presentation to Attending
31-year-old with positive pregnancy test, mild lower abdominal pain, and scant vaginal spotting, hemodynamically stable on arrival. Transvaginal ultrasound did not show a definite intrauterine pregnancy or ectopic, so this is a pregnancy of unknown location. She has no peritoneal signs, no shoulder pain, no syncope, and her abdomen is soft with only mild suprapubic tenderness. I’ve sent CBC, type and screen, quantitative hCG, and Rh testing, and I’m planning Rh immune globulin if she’s Rh-negative, plus strict ectopic precautions and 48-hour repeat hCG/TVUS follow-up.
Study Directive
Draw the PUL pathway from memory: positive test → nondiagnostic TVUS → quantify hCG → repeat hCG/US → classify outcome.
Practice one sentence explaining why a nondiagnostic scan cannot exclude ectopic pregnancy.
Memorize RhIg indications and doses for early pregnancy bleeding.
Review how hCG trends relate to ultrasound visibility without overcommitting to a single cutoff.
Key Medications
Normal saline or LR: 1–2 L IV
Rh(D) immune globulin: 50 mcg IM once in early pregnancy bleeding/ectopic concern; 300 mcg IM if 50 mcg not available or per protocol
Acetaminophen: 650–1000 mg PO/IV
Analgesic or antiemetic choices vary; if uncertain in early pregnancy, check institutional/OB guidance.
Methotrexate dosing for confirmed ectopic is protocol-dependent and not an ED guessing game — verify with OB/GYN and reference sources.
High-Yield Pearls
“Unknown location” is a temporary label, not a destination.
Instability beats ultrasound: a crashing pregnant patient is ectopic until proven otherwise, even if the scan is nondiagnostic.
Follow-up reliability is part of risk stratification; a stable patient who cannot return safely may need observation/admission.
The Mimics
Very early viable intrauterine pregnancy — the single discriminator is time and serial imaging; confusing it with ectopic can lead to unnecessary intervention.
Early pregnancy loss — falling hCG and bleeding/cramping dominate; missing ectopic in this pathway still risks rupture.
Corpora lutea / adnexal cyst pain — localized pain without worsening shock; confusing it with ectopic can trigger unnecessary surgery, but it must be distinguished carefully.
Non-gynecologic abdominal pain — appendicitis/renal colic can coexist with pregnancy; mislabeling it ectopic delays the real source.
Board Question
A 29-year-old with positive pregnancy test and mild vaginal bleeding has an ultrasound that shows no intrauterine pregnancy and no adnexal mass. She is stable, with benign abdominal exam. What is the best next step?
AReassure and discharge without follow-up
BDiagnose ectopic pregnancy and give methotrexate
CTreat as pregnancy of unknown location with serial hCG and repeat ultrasound
DSend her immediately to the operating room
Reveal answer
Correct: C
A positive test with nondiagnostic ultrasound is a pregnancy of unknown location, not a ruled-out ectopic or confirmed IUP. Stable patients need serial quantitative hCG and repeat imaging with clear return precautions; methotrexate is reserved for confirmed management pathways, not guesswork.
A practical guideline for ED disposition of stable PUL, emphasizing serial β-hCG, repeat transvaginal ultrasound, ectopic precautions, and urgent intervention when instability or concerning ectopic findings are present.
3 of 4
Postpartum Hemorrhage
Postpartum hemorrhage can kill in minutes. The difference between controlled bleeding and exsanguination is rapid recognition, uterine tone management, and...
A 33-year-old who delivered 20 minutes ago is suddenly pale, soaked through two chux pads, and staring past you while a nurse says, “I think it’s still bleeding.” The fundus feels soft above the umbilicus, and the room has that fast, quiet panic of everyone counting sponges without saying it. Her blood pressure is drifting down, and the stretcher is turning red at the edges. You have seconds to decide whether this is tone, tissue, trauma, or clot.
Before You Read
What are the four Ts, and which one is most common?
What do you do in the first fifteen minutes when postpartum bleeding is not slowing?
When do you escalate to blood products, procedures, and the OR?
Why It Matters
Postpartum hemorrhage can kill in minutes. The difference between controlled bleeding and exsanguination is rapid recognition, uterine tone management, and a standardized escalation sequence.
When to Think of It
Think of PPH with heavy vaginal bleeding after delivery, soaked pads/chux, clots, uterine bogginess, tachycardia, hypotension, or failure of the uterus to contract. It can be immediate or delayed; any unstable postpartum patient with bleeding is PPH until proven otherwise.
Sick or Not Sick
The key fork is stable, responding to uterotonics vs. ongoing hemorrhage/shock despite initial measures. If she is unstable, you activate hemorrhage response and treat while searching for the source, not after.
The First Fifteen Minutes
Call for help: OB, anesthesia, blood bank, nursing hemorrhage protocol.
Bimanual uterine massage and fundal massage immediately, because tone is the commonest cause and mechanical stimulation can compress bleeding vessels.
Oxytocin 10 units IM or 10–40 units IV infusion (per institutional protocol) now, because it is first-line uterotonic and improves contraction.
Tranexamic acid 1 g IV over 10 minutes as early as possible, ideally within 3 hours of birth; may repeat 1 g IV once if bleeding continues after 30 minutes or recurs, because it inhibits fibrinolysis and reduces death from bleeding.
If hemorrhage persists and no contraindication:
Methylergonovine 0.2 mg IM, repeat every 2–4 hours if needed, avoid in hypertension/preeclampsia because it causes vasoconstriction and uterine contraction.
Carboprost 250 mcg IM, repeat every 15–90 minutes up to 8 doses, avoid in asthma because it is a prostaglandin F2α uterotonic that can provoke bronchospasm.
Misoprostol 800–1000 mcg PR/SL/PO once, because prostaglandin activity augments uterine tone.
Two large-bore IVs, CBC, coags, fibrinogen, type & cross, and rapid transfusion if unstable, because volume and clot factor replacement are often required.
Crystalloid while blood is coming, but do not chase hemorrhage with liters of saline alone; it dilutes clotting factors.
Definitive Care & Disposition
Identify the cause: Tone (uterine atony), Tissue (retained placenta/products), Trauma (laceration, inversion, rupture), Thrombin (coagulopathy). Retained tissue needs placental/manual extraction and OB intervention. Lacerations or uterine inversion need procedural/surgical management. Ongoing bleeding or hemodynamic instability requires massive transfusion protocol, higher-level monitoring, and likely OR/IR. Admit all significant PPH; discharge is not the correct endpoint.
How This One Kills
The fatal miss is assuming the uterus will “clamp down on its own” while bleeding continues. Delayed recognition of retained placenta, laceration, or coagulopathy turns a fixable hemorrhage into DIC and shock.
The Atypical Presentation
Not every PPH announces itself with a torrent. Some patients bleed steadily under drapes, saturate pads more slowly, or only look “a little tachycardic” until they suddenly are not. A firm uterus with continued bleeding should push you away from atony and toward trauma or tissue, while a soft, enlarged fundus points back to tone. In older, exhausted, or heavily anesthetized patients, vital sign changes may lag behind the actual blood loss.
Back to Our Patient
Back to our patient: the 33-year-old who is soaking chux 20 minutes after delivery has postpartum hemorrhage, most likely uterine atony if the fundus is soft. You mobilize the hemorrhage response, start fundal massage, give oxytocin, add TXA, and escalate uterotonics based on contraindications while sending blood and checking for laceration, retained tissue, or inversion. If the bleeding does not rapidly improve or she becomes unstable, she needs immediate OB-led procedural management, likely transfusion, and higher-level care.
Patient Presentation to Attending
33-year-old immediately postpartum with heavy vaginal bleeding and pallor, now tachycardic with downtrending blood pressure. Nursing reports soaked chux and large clots; the fundus is boggy above the umbilicus. She has no fever and no obvious laceration on quick inspection, but bleeding is ongoing, so I’m activating postpartum hemorrhage protocol. I’ve started bimanual massage, ordered oxytocin and TXA, sent CBC/coags/type and cross, and I’m calling OB and blood bank now for definitive management and transfusion support.
Study Directive
Rehearse the “four Ts” and match each to a bedside clue.
Memorize first-line uterotonics, doses, and contraindications.
Practice a 60-second hemorrhage activation presentation to OB and blood bank.
Draw a one-page algorithm for postpartum bleeding from massage to OR.
Key Medications
Oxytocin: 10 units IM once or 10–40 units IV infusion
Tranexamic acid: 1 g IV over 10 min; may repeat 1 g IV once
Methylergonovine: 0.2 mg IM q2–4h PRN; avoid HTN/preeclampsia
Carboprost: 250 mcg IM q15–90 min PRN, max 8 doses; avoid asthma
Misoprostol: 800–1000 mcg PR/SL/PO once
Blood products per hemorrhage protocol; dosing is protocol-specific
Doses may vary by institution and OB protocol; verify if uncertain. Pediatric differences are not relevant.
High-Yield Pearls
A boggy uterus = tone until proven otherwise; a firm uterus with bleeding = think trauma or tissue.
TXA works best early in postpartum hemorrhage; don’t wait for the hemorrhage to become “severe enough.”
Asthma and hypertension matter because they determine which uterotonics you can safely use.
The Mimics
Uterine atony — boggy fundus and diffuse bleeding; confusing it with laceration delays the correct procedural fix.
Retained products/placenta — placenta incomplete or bleeding persists despite firm uterus; missing it prevents definitive evacuation.
Genital tract laceration — firm uterus but brisk bleeding; mistaking it for atony leads to ineffective uterotonics alone.
Placental inversion — severe pain, shock, mass at introitus/uterus absent in fundus; missing it can be catastrophic during attempted fundal massage.
Board Question
A postpartum patient has heavy vaginal bleeding, a soft uterus, and tachycardia. What is the best immediate medication in addition to uterine massage?
ACeftriaxone
BOxytocin
CMagnesium sulfate
DCarbamazepine
Reveal answer
Correct: B
Uterine atony is the most common cause of postpartum hemorrhage, and oxytocin is the first-line uterotonic. Antibiotics or anticonvulsants do not address the bleeding source.
Provides a practical, up-to-date framework for ED and obstetric teams managing postpartum hemorrhage with shock: quantify blood loss, activate massive transfusion early, give uterotonics and tranexamic acid promptly, and escalate to tampona
4 of 4
Endometritis
Endometritis is a common postpartum infection and a classic cause of fever, uterine tenderness, and maternal morbidity. Missing it delays antibiotics and...
A 29-year-old who delivered three days ago winces every time the wheelchair hits a crack in the pavement. She smells faintly of lochia, is holding her lower abdomen, and says the pain is “deeper” than cramping. At triage she looks flushed, with a temp of 38.7°C and a heart rate that stays elevated even after she sits. The incision is not the issue, the bleeding is not the issue, and the uterus is tender when you press — but you still need to separate infection from the other postpartum threats.
Before You Read
What is the classic postpartum endometritis picture?
Which postpartum patient with fever is sick enough to need IV antibiotics and admission?
What alternative diagnoses must you exclude when the uterus is tender?
Why It Matters
Endometritis is a common postpartum infection and a classic cause of fever, uterine tenderness, and maternal morbidity. Missing it delays antibiotics and can allow progression to sepsis or pelvic abscess.
When to Think of It
Think endometritis in postpartum patients — especially after C-section, prolonged rupture of membranes, prolonged labor, or multiple vaginal exams — who have fever, uterine fundal tenderness, foul-smelling lochia, chills, and lower abdominal pain. It can present with malaise and tachycardia even before dramatic findings.
Sick or Not Sick
The key fork is toxic/septic/unstable vs. localized postpartum infection with reassuring vitals. Fever plus uterine tenderness after delivery is usually enough to treat; hypotension, altered mentation, or lactate elevation means sepsis pathway and admission.
The First Fifteen Minutes
CBC, lactate if ill, blood cultures if septic-appearing, and pelvic/abdominal exam.
IV fluids 1–2 L crystalloid if tachycardic or septic, because perfusion supports early antibiotic delivery.
Clindamycin 900 mg IV every 8 hours + gentamicin 5 mg/kg IV every 24 hours for typical postpartum endometritis, because broad anaerobic/aerobic coverage targets polymicrobial uterine infection.
If gentamicin is not ideal or local protocol differs, some regimens use ampicillin-sulbactam 3 g IV every 6 hours; check local guidance.
Acetaminophen 650–1000 mg PO/IV for fever/pain, because symptom control helps but does not replace antibiotics.
If concern for retained products or pelvic abscess, OB consultation now; antibiotics alone may not be enough.
Definitive Care & Disposition
Most postpartum endometritis needs admission for IV antibiotics until clinically improved and afebrile, then transition per OB. If fever persists beyond 48–72 hours on appropriate therapy, think retained products, wound infection, septic pelvic thrombophlebitis, or abscess and escalate imaging/consultation. Post-cesarean patients, toxic patients, or anyone with unstable vitals deserves inpatient care; discharge is uncommon.
How This One Kills
The dangerous miss is calling postpartum fever “just mastitis, just UTI, or just normal lochia” and not treating the uterine source. The other miss is missing retained products or another pelvic infection when fever does not improve on antibiotics.
The Atypical Presentation
Endometritis is not always a screaming septic patient. Some postpartum patients present with just malaise, low-grade fever, uterine soreness, and a “bad smell” complaint that gets minimized, especially after a C-section or prolonged labor. The sign that should keep you alert is a tender uterus in the postpartum window, particularly when the fever lacks a better explanation and the patient feels progressively worse instead of better.
Back to Our Patient
Back to our patient: the 29-year-old on postpartum day 3 with fever, uterine tenderness, and foul lochia has postpartum endometritis. She is not a discharge candidate because she is febrile and tachycardic, so you start IV fluids, send labs, and give clindamycin plus gentamicin or a local equivalent regimen. If she improves as expected, she stays inpatient until afebrile and clinically better; if she does not, you escalate for retained products, abscess, or septic pelvic thrombophlebitis.
Patient Presentation to Attending
29-year-old postpartum day 3 after vaginal delivery with fever, worsening lower abdominal pain, and foul-smelling lochia. She is tachycardic, febrile to 38.7, and has uterine fundal tenderness on exam without breast symptoms or flank pain. I’m concerned for postpartum endometritis rather than mastitis or pyelonephritis, and I’ve ordered CBC and cultures if needed, started IV fluids, and am giving clindamycin plus gentamicin while consulting OB for admission. If she fails to improve, we’ll look for retained products or a pelvic abscess.
Study Directive
Memorize the classic postpartum endometritis triad and common risk factors.
Practice choosing the correct antibiotic regimen from memory.
Compare endometritis vs mastitis vs pyelonephritis in a one-minute verbal differential.
Review when persistent fever after treatment demands imaging and OB re-evaluation.
Mechanism Pearl of the Day: Across these topics, the recurring mechanism is failure of a protective barrier to contain a dangerous process: vomiting starves and dehydrates, a pregnancy that is not yet localized can rupture silently, uterine atony fails to compress placental vessels, and postpartum infection exploits the open uterine cavity. The bedside skill is spotting when physiology has crossed from expected change into loss of containment.
Key Medications
Clindamycin: 900 mg IV q8h
Gentamicin: 5 mg/kg IV q24h
Ampicillin-sulbactam: 3 g IV q6h as an alternative regimen depending on protocol
Acetaminophen: 650–1000 mg PO/IV
Dosing may vary by institutional postpartum infection protocol and renal function; check reference sources if uncertain. For gentamicin, use actual/adjusted body weight per pharmacy protocol.
High-Yield Pearls
Postpartum fever + uterine tenderness = endometritis until proven otherwise, especially after C-section.
Failure to improve in 48–72 hours is a source-control problem, not just an antibiotic-tuning problem.
Foul lochia is a clue, not a diagnosis; always reassess for retained products or abscess if the course is atypical.
The Mimics
Mastitis — breast pain, erythema, focal breast tenderness; confusing it with endometritis delays the correct pelvic workup.
Pyelonephritis — flank pain, urinary symptoms, CVA tenderness; missing it delays renal-source treatment and sepsis care.
Septic pelvic thrombophlebitis — persistent fever despite antibiotics; confusing it with routine endometritis delays anticoagulation/advanced workup.
Retained products of conception — ongoing bleeding/subinvolution and fever; missing it prevents source control.
Board Question
A postpartum patient has fever, uterine tenderness, and foul-smelling lochia 4 days after cesarean delivery. Which is the best empiric treatment?
AClindamycin plus gentamicin
BOral azithromycin alone
CVancomycin alone
DNo antibiotics; this is normal postpartum change
Reveal answer
Correct: A
Postpartum endometritis is typically polymicrobial and needs broad IV coverage, classically clindamycin plus gentamicin. Oral monotherapy or observation alone is inadequate when uterine infection is suspected.
Yesterday’s Differential
The daily puzzle — from editions past
A quick test of recall from prior editions. Commit to an answer before you check.
From yesterday's edition
EMS rolls in doing compressions. The monitor shows a chaotic irregular waveform without identifiable QRS complexes. What’s the diagnosis, and the first move?
Check your answer
Ventricular Fibrillation. Defibrillate, resume CPR immediately, follow ACLS shockable rhythm algorithm, and search for reversible causes during rhythm checks and after ROSC.
From the July 1 edition
Today, three days ago: Magnesium Sulfate. What’s the adult ED dose, and the contraindication you’d most regret missing?
Check your answer
Torsades: 2 g IV over 1–2 min, may repeat. Severe asthma: 2 g IV over 20 min. Eclampsia: 4–6 g IV load over 15–20 min, then 1–2 g/h infusion. Heart block; myasthenia gravis (relative); severe renal impairment without level monitoring.
From the June 24 edition
A 28-year-old woman presents with 4 days of progressive ascending weakness after a viral gastroenteritis. She has areflexia, mild distal paresthesias, and difficulty taking a deep breath. Which is the most appropriate next step?
AHigh-dose IV methylprednisolone
BSerum acetylcholine receptor antibodies and discharge if normal
CSerial FVC/NIF measurements and ICU admission
DEmergent lumbar puncture must confirm the diagnosis before treatment
Reveal answer
Correct · C
Guillain-Barré syndrome is a clinical diagnosis in the ED, and the immediate threat is respiratory failure. Serial respiratory mechanics and ICU-level monitoring are essential; steroids do not help, and CSF confirmation should not delay disposition or treatment.
Journal Watch
From the FOAMed wire
Notable posts and reviews from the last week, ranked by relevance to today’s lead and source trust.
Journal Feed covers pediatric seizure treatment, BB and CCB overdose management, and EVT for medium vessel strokes. The post Journal Feed Weekly Wrap-Up appeared first on emDocs .
Podcast Picks
Two for the shift
Critical Care Perspectives in Emergency Medicine2026-05-23
Acute hypoxemic respiratory failure is a leading cause of ICU admission worldwide. Oxygen is first-line therapy for patients with acute hypoxemic respiratory failure and can be given via nasal cannula (NC), non-rebreather mask (NRB), high-flow nasal cannula (HFNC), or noninvasive ventilation (NIV). At present, the literature is inconsistent on which mode...
Source
Critical Care Perspectives in Emergency Medicine
Published
2026-05-23
Host
Critical Care Perspectives in Emergency Medicine, Critical Care Perspectives in Emergency Medicine
Facial trauma is common in emergency medicine, but the biggest pitfalls are often not the fractures themselves—they're the threatened airway, vision-threatening ocular injuries, missed septal hematomas, and subtle...
UMEM Pearl
Matched to today’s topics
A clinical pearl from the University of Maryland EM group’s Educational Pearls, tied to today’s differential.
In pregnancy of unknown location, hypotension without tachycardia is not reassuring: ruptured ectopic with hemoperitoneum can present with paradoxical bradycardia and should be resuscitated and worked up like any other hemorrhagic shock.
Bradycardia accompanying hypotension can be found in spinal cord injury (loss of autonomic reflex), beta blocker and calcium channel blocker overdose, intrinsic cardiac electrophysiologic derangement, and, often forgotten, intrabdominal hemorrhage. In the appropriate setting (blunt trauma, ruptured ectopic pregnancy), bradycardic hypotensive patients should be considered the same as tachycardic hypotensive patients and get a work up and treatment focused on Hemoperitoneum.
Critical Care Corner
Matched to today’s topics
A critical-care reference from LITFL’s Critical Care Compendium, tied to today’s differential.
For postpartum hemorrhage, the ICU lens is physiology: a term uterus can siphon massive flow, so visible blood loss and maternal vital signs may lag behind the pace of decompensation.
Hemorrhage and Pregnancy: gravid uterus receives 15% of Q -> bleeding can be rapid; principle causes; abruption, placenta praevia, PPH; baby more at risk than mum
Pharmacology Corner
Two drugs for the shift
One antimicrobial and one other ED workhorse — selected daily, with sources and last-reviewed dates so every dose is cross-checkable.
Antimicrobial of the Day
Ampicillin
Aminopenicillin (IV)
Indication
Listeria coverage in meningitis at the extremes of age and in immunocompromise, enterococcal infections, GBS, neonatal sepsis, susceptible UTI, and enterococcal endocarditis (combination therapy).
What’s your dose? — reveal dosing & cautions
ED Dose
2 g IV q4h for meningitis/Listeria. Enterococcal endocarditis: 2 g IV q4h (with ceftriaxone or gentamicin). Neonatal sepsis: weight/age-based.
Renal Adjustment
Extend dosing interval when CrCl < 30 mL/min.
Contraindications
Penicillin hypersensitivity.
Interactions
Allopurinol increases rash incidence; may reduce efficacy of oral contraceptives.
Monitoring
Allergy/anaphylaxis, renal function, rash.
ED Pearl
Add ampicillin to empiric meningitis coverage at the extremes of age (neonates, > 50 years) and in immunocompromise to cover Listeria — ceftriaxone plus vancomycin misses it.
Acute agitation, nausea/vomiting, migraine adjunct, cannabinoid hyperemesis, and vertigo-associated nausea in selected patients.
What’s your dose? — reveal dosing & cautions
ED Dose
Nausea/migraine: 0.625–2.5 mg IV/IM. Agitation: 5–10 mg IM/IV depending on severity and local protocol.
Renal Adjustment
No renal adjustment; caution in hepatic impairment.
Contraindications
Known QT prolongation/TdP risk when significant, Parkinson disease/Lewy body dementia caution, severe CNS depression.
Interactions
Other QT-prolonging drugs, CNS depressants, dopamine agonists antagonized.
Monitoring
Sedation, BP, extrapyramidal symptoms, QTc in high-risk patients or higher/repeated doses.
ED Pearl
Droperidol is one of the most useful ED symptom-control drugs; respect QT risk, but do not let the black-box warning erase good medicine in appropriately selected patients.
For educational use only. Verify dosing against the FDA label and your institution’s pharmacy resources before administering.
ECG of the Day
Channelopathy
Long QT Syndrome
QT prolongation is a substrate for torsades; the ED job is to identify the trigger before the malignant rhythm appears.
The Tracing
A 36-year-old woman presents after syncope while taking escitalopram, hydroxyzine, and azithromycin for a respiratory illness. Her ECG shows a QTc over 560 ms with broad-based T waves and a pause-dependent premature ventricular beat. Potassium returns at 3.1 mmol/L and magnesium is low. She is currently awake.
QTc prolongation, commonly >470 ms in men or >480 ms in women and especially concerning when >500 ms
May show broad-based T waves, T-wave notching, prominent U waves, or T-U fusion depending on cause
Pause-dependent PVCs can initiate torsades
Acquired causes include medications, hypokalemia, hypomagnesemia, hypocalcemia, bradycardia, and structural heart disease
Inherited forms include Romano-Ward and Jervell-Lange-Nielsen variants
Pearls
QTc >500 ms is the threshold where your attention should sharpen; risk rises further with hypokalemia, bradycardia, and medication stacking.
Medication reconciliation is treatment. Stop the offending drugs and correct potassium and magnesium aggressively.
Syncope with prolonged QT is not benign fainting; it may be self-terminating torsades.
Pitfalls
Automated QTc can be wrong when T and U waves merge. Manually inspect the tracing when the number drives disposition.
Do not give more QT-prolonging antiemetics or antipsychotics to the patient whose ECG is already warning you.
Congenital long QT may have a normal-looking ECG at times; family history and exertional or auditory-trigger syncope matter.
At the Bedside
Stop QT-prolonging medications, correct K/Mg/Ca, place on telemetry, and treat torsades immediately if it occurs. Syncope, QTc >500 ms with symptoms, or congenital concern warrants admission or urgent cardiology input.
For educational use only. Verify ECG interpretation against the LITFL entry and your institution’s practice before clinical decision-making.
Case of the Day
From the lead · Nausea, Vomiting and Hyperemesis in Pregnancy
Self-Examination
Test Your Understanding
A 24-year-old at 9 weeks’ gestation presents with 5 days of vomiting and inability to tolerate PO. She is tachycardic, orthostatic, and has ketonuria. What is the best next step?
AReassure her that this is normal morning sickness
BGive dextrose-containing IV fluids immediately
CGive thiamine, IV fluids, and an antiemetic
DDischarge with antibiotics for presumed gastroenteritis
Reveal answer
Correct answer · C
This is hyperemesis gravidarum with dehydration/starvation. Thiamine should be given before dextrose if poor intake is prolonged, and IV fluids plus antiemetics are the immediate management. Normal morning sickness is not associated with ketonuria and orthostasis.
Study Pace4 topics today; 40 remaining; Day 33 of 43Deadline · June 1, 2026