An Emergency Medicine Broadsheet
·Phoenix·
Est. MMXXVI
Blue Fish Med · Today's Topic
Shoulder Dystocia
A shoulder dystocia can turn a routine birth into a time-critical emergency with fetal hypoxia, brachial plexus injury, or maternal hemorrhage. Coordinated maneuvers matter more than force.
Also known asimpacted shoulder · fetal shoulder dystocia
The baby’s head delivers, then draws tightly back against the perineum. The room’s conversation stops as the nurse says the shoulders have not followed. The fetal monitor still sounds, but the next contraction is already building. Which move comes first?
— What’s your move? Read on.
Before you read
When should you move on from the initial maneuver?
What findings change the newborn and maternal priorities afterward?
When to Think of It
The head delivers but the shoulders do not with gentle axial traction; the head may retract against the perineum (“turtle sign”). Call shoulder dystocia when the shoulders fail to deliver promptly with routine gentle traction—not only after a fixed time has elapsed.
Sick or Not Sick
The key call is whether the shoulder is still impacted after the first maneuver. Announce the emergency, summon experienced obstetric, neonatal, and anesthesia help, and track time while moving through maneuvers without forceful traction.
The First Fifteen Minutes
Immediately: Call for help, stop pushing if directed, and begin McRoberts positioning—hyperflex the mother’s hips; add suprapubic pressure directed to dislodge the anterior shoulder. Do not apply fundal pressure or use excessive downward traction.
If unresolved, proceed promptly to internal rotational maneuvers (e.g., Rubin/Woods) or deliver the posterior arm; choose based on operator skill and fetal position. Move to hands-and-knees positioning if feasible.
Refractory dystocia: Consider terbutaline 0.25 mg subcutaneously once only if uterine relaxation may facilitate a maneuver; it is an adjunct, not a substitute for maneuvers, and may cause maternal tachycardia.
If the shoulders remain impacted despite appropriate maneuvers, the senior obstetrician directs rescue options; do not improvise forceful traction. Prepare neonatal resuscitation.
Definitive Care & Disposition
After delivery, assess the newborn promptly for respiratory compromise, clavicle or humeral injury, and brachial plexus dysfunction. Examine the mother for major lacerations and postpartum hemorrhage; quantify blood loss and manage complications. Document the sequence, timing, maneuvers, personnel, and maternal/newborn outcomes.
How This One Kills
Repeated downward traction or fundal pressure can worsen impaction and cause serious fetal injury or uterine rupture. The failure mode is escalating force instead of changing the mechanics.
The Differential — What Else Looks Like This
Nuchal cord — the cord is around the neck and the shoulders are not impacted; confusing it can delay the needed shoulder maneuvers.
Breech delivery difficulty — the body delivers before the head; the mechanics and rescue maneuvers differ, so misclassification wastes critical time.
The Second-Day Story
Dystocia may be recognized without a dramatic turtle sign, especially when the head delivers slowly or the shoulder obstruction is not immediately obvious. Do not wait for a particular visual cue or elapsed-time threshold: if routine gentle traction does not deliver the shoulders, call the emergency and begin the maneuver sequence.
Back to Our Patient
Back to our patient. The delivered head retracts tightly against the perineum, and gentle axial traction does not deliver the shoulders: recognize shoulder dystocia, call for help, and move immediately to McRoberts positioning with suprapubic pressure. When that does not resolve the impaction, the obstetrician proceeds to an internal rotational maneuver and delivers the baby without forceful traction. The neonatal team assesses the newborn, while the obstetric team checks the mother for trauma and hemorrhage; both remain for appropriate post-delivery observation.
Patient Presentation to Attending
How you’d present this patient on the floor — tight, pertinent positives and negatives, no rambling
“This is a term laboring patient with a vaginal delivery complicated by failure of the shoulders to deliver after the head emerged. The head has retracted against the perineum, and gentle axial traction has not delivered the shoulders. There is no evidence of a nuchal cord explaining the delay, and the fetal heart rate remains audible. I called the shoulder dystocia, stopped traction, and started McRoberts positioning with suprapubic pressure; the dystocia persists. I recommend proceeding immediately to an internal rotational maneuver or posterior-arm delivery with the senior obstetrician, while neonatal and hemorrhage teams prepare.”
Study Directive
From memory, list the first three shoulder dystocia maneuvers in order and name one rescue maneuver.
Practice a 20-second team callout that identifies the emergency, requests help, and assigns a timekeeper.
Review your institution’s shoulder dystocia documentation and newborn-examination process.
Use a stepwise sequence of delivery maneuvers when shoulder dystocia occurs, while avoiding excessive traction on the fetal head.
More in Today's Issue
5 additional topics
2 of 6
Chest Pain in Pregnancy
Pregnancy and the postpartum period increase risk for thromboembolism, acute coronary syndromes, and aortic disease. Delayed diagnosis or withholding...
At 29 weeks’ gestation, 31-year-old Maya grips the edge of the stretcher as a breath catches short. Her chest pain sharpens when she inhales; one calf is more swollen than the other. Her blood pressure is steady, and the first ECG is nondiagnostic. Which dangerous cause should be excluded first?
Before You Read
Which pregnancy-adapted approach can help decide whether pulmonary vascular imaging is needed?
Which findings make this an unstable patient rather than a diagnostic-workup patient?
When should pregnancy change your imaging or treatment plan—and when should it not?
Why It Matters
Pregnancy and the postpartum period increase risk for thromboembolism, acute coronary syndromes, and aortic disease. Delayed diagnosis or withholding indicated imaging can be more dangerous than the radiation exposure.
When to Think of It
Treat new chest pain, dyspnea, syncope, hypoxemia, or unexplained tachycardia in pregnancy or postpartum as potentially serious. Consider pulmonary embolism (PE), acute coronary syndrome (ACS), aortic dissection, pneumothorax, and peripartum cardiomyopathy—not just reflux or musculoskeletal pain.
Sick or Not Sick
The crucial fork is unstable versus stable. Shock, severe hypoxemia, ongoing ischemic symptoms, malignant arrhythmia, or suspected dissection demands resuscitation and immediate targeted evaluation; a stable patient can undergo a structured, diagnosis-directed workup.
The First Fifteen Minutes
Obtain ECG, serial high-sensitivity troponins, pulse oximetry, focused history/exam, and bedside cardiac/lung assessment as indicated. Give oxygen only for hypoxemia; do not delay chest radiography or definitive imaging solely because of pregnancy.
For suspected ACS, give aspirin 162–325 mg PO, chewed once, unless contraindicated; it inhibits platelet aggregation. For ongoing ischemic pain with adequate blood pressure and no contraindication, give nitroglycerin 0.4 mg SL every 5 minutes, up to 3 doses; it reduces preload and myocardial oxygen demand. If treating ACS with anticoagulation, use unfractionated heparin 60 units/kg IV (maximum 4,000 units), then 12 units/kg/hour IV (maximum 1,000 units/hour) per local ACS protocol; it limits thrombus propagation. Avoid nitrates with hypotension or recent phosphodiesterase-5 inhibitor use.
For suspected PE with high clinical concern and delayed imaging, if no major bleeding contraindication, start enoxaparin 1 mg/kg SC every 12 hours; it provides therapeutic anticoagulation. Do not anticoagulate reflexively if aortic dissection is a serious possibility.
Definitive Care & Disposition
In a stable patient with possible PE, consider pregnancy-adapted YEARS: assess signs of DVT, hemoptysis, and whether PE is the most likely diagnosis. With no YEARS criteria, a D-dimer below 1,000 ng/mL may exclude PE; with one or more criteria, the threshold is below 500 ng/mL (use the local assay and units). If not excluded, proceed to compression ultrasound when DVT symptoms are present and to appropriate chest imaging—V/Q scan or CT pulmonary angiography based on the clinical context and local pathway. For suspected ACS, involve cardiology and obstetrics early; pursue indicated angiography/reperfusion. Suspected dissection requires urgent definitive imaging and specialist care. Admit patients with confirmed or strongly suspected life-threatening disease; disposition depends on diagnosis, stability, and gestational age.
How This One Kills
Anchoring on pregnancy-related reflux, anxiety, or “normal” pregnancy tachycardia can delay recognition of PE, ACS, or dissection. Pregnancy is not a reason to withhold necessary imaging or treatment.
The Atypical Presentation
Pregnant and postpartum patients may have nonspecific dyspnea, fatigue, nausea, or mild tachycardia, while normal pregnancy itself can raise heart rate and D-dimer. Do not use a single symptom, normal oxygen saturation, or a nondiagnostic ECG to dismiss serious disease. Reassess the whole pattern, use a validated pregnancy-specific pathway where appropriate, and obtain definitive testing when indicated.
Back to Our Patient
Back to 31-year-old Maya, whose pleuritic pain and unilateral calf swelling make PE a leading concern despite stable vital signs and a nondiagnostic ECG. Recognize the possible PE, risk-stratify her as stable, and assess the DVT signs and other pregnancy-adapted YEARS items. Her symptoms include a clinical DVT sign, so the team obtains urgent compression ultrasound and proceeds to chest imaging when needed rather than dismissing the symptoms or delaying for pregnancy. With PE confirmed and no bleeding contraindication, she receives weight-based therapeutic anticoagulation and is admitted for obstetric and medical management.
Patient Presentation to Attending
“This is a 31-year-old at 29 weeks’ gestation with acute pleuritic chest pain and dyspnea, plus unilateral calf swelling. She is normotensive and not hypoxemic; ECG is nondiagnostic, and there is no ongoing ischemic pattern or reported abrupt tearing pain. The calf asymmetry raises concern for DVT and PE, and pregnancy does not lower that concern. I recommend pregnancy-adapted PE evaluation with compression ultrasound and definitive chest imaging if needed. If PE is confirmed and there is no bleeding contraindication, I will start therapeutic enoxaparin and admit with obstetric involvement.”
Study Directive
Write the three pregnancy-adapted YEARS items and both D-dimer thresholds from memory.
Practice an oral differential for pregnancy-associated chest pain that includes PE, ACS, dissection, and peripartum cardiomyopathy.
Review your local pathway for choosing V/Q scanning versus CT pulmonary angiography.
Key Medications
Aspirin: 162–325 mg PO, chewed once for suspected ACS if not contraindicated.
Nitroglycerin: 0.4 mg SL every 5 minutes, up to 3 doses for ongoing ischemic pain if blood pressure permits.
Unfractionated heparin (ACS): 60 units/kg IV bolus (maximum 4,000 units), then 12 units/kg/hour IV (maximum 1,000 units/hour); follow institutional protocol.
Enoxaparin (therapeutic PE): 1 mg/kg SC every 12 hours; adjust for renal function and follow local protocol. Pregnancy-specific anticoagulation decisions merit obstetric/hematology input.
High-Yield Pearls
Pregnancy-adapted YEARS uses different D-dimer thresholds depending on whether any YEARS criteria are present; do not apply a single cutoff indiscriminately.
A normal oxygen saturation or nondiagnostic ECG does not rule out PE.
The Mimics
GERD — burning discomfort linked to meals or recumbency without cardiopulmonary compromise; mislabeling ischemia or PE as reflux delays definitive care.
Musculoskeletal chest pain — reproducible focal tenderness supports the diagnosis but does not by itself exclude PE or ACS; premature closure is the danger.
Peripartum cardiomyopathy — heart-failure symptoms and reduced LV function distinguish it from isolated chest-wall pain; missing it risks rapid decompensation.
Aortic dissection — abrupt severe pain, pulse/BP differential, or neurologic deficit should redirect evaluation urgently; anticoagulating presumed PE before considering dissection can be catastrophic.
Board Question
A hemodynamically stable pregnant patient has pleuritic chest pain, unilateral calf swelling, and suspected PE. Imaging is not immediately available, and there is no major bleeding contraindication or concern for aortic dissection. Which treatment is appropriate while arranging definitive evaluation? -
AWithhold anticoagulation until after delivery -
BEnoxaparin 1 mg/kg SC every 12 hours -
CAspirin 81 mg PO daily as sole treatment -
DWarfarin loading dose
Reveal answer
Correct: B
Therapeutic low-molecular-weight heparin is an appropriate treatment for suspected or confirmed PE in pregnancy when anticoagulation is indicated. Warfarin is avoided during pregnancy, and aspirin is not adequate treatment for PE.
Use a pregnancy-specific approach to recognize and manage dangerous cardiac causes of chest pain, including acute coronary syndrome and aortic dissection.
For pregnant patients with chest pain and suspected pulmonary embolism, the pregnancy-adapted YEARS pathway helps guide D-dimer testing and decisions about chest imaging.
Toxic shock syndrome (TSS) can progress rapidly to refractory shock and multiorgan failure. Early resuscitation, toxin-suppressing antimicrobials, and...
Twenty-four-year-old Elena arrives flushed and shivering, her blood pressure low despite a rapid first liter of fluid. A red rash spreads across her trunk, and she says she has felt suddenly ill since yesterday. The source of the illness is not yet clear. What needs to happen before the full workup is complete?
Before You Read
Which bedside finding changes this from a rash-and-fever workup to a resuscitation?
Why should treatment cover both toxin-producing staphylococci and invasive streptococci at first?
What must be done to control the source—not just the blood pressure?
Why It Matters
Toxic shock syndrome (TSS) can progress rapidly to refractory shock and multiorgan failure. Early resuscitation, toxin-suppressing antimicrobials, and source control are time-critical.
When to Think of It
Consider TSS with abrupt fever, diffuse erythematous rash, hypotension, and systemic illness—especially with menstruation, a retained tampon or other foreign body, a wound, recent surgery, or postpartum infection. Desquamation may appear later and is not required to begin treatment.
Sick or Not Sick
The key call is whether the patient has shock or organ dysfunction. Hypotension, altered mentation, poor perfusion, or suspected invasive infection warrants immediate resuscitation and ICU-level evaluation; do not wait for culture results or late diagnostic criteria.
The First Fifteen Minutes
Establish monitoring and IV access, obtain cultures if this does not delay antibiotics, check lactate and organ-function studies, and rapidly inspect for removable foreign material or a drainable/infected source. For shock, give isotonic crystalloid in reassessed boluses toward 30 mL/kg; stop or modify for pulmonary edema or other evidence of overload.
For suspected TSS with shock, start vancomycin 20–25 mg/kg IV loading dose (maximum 3,000 mg; use institutional protocol) plus clindamycin 900 mg IV every 8 hours for empiric coverage and toxin suppression, and piperacillin–tazobactam 4.5 g IV every 6–8 hours for broad source coverage. Do not delay antibiotics for cultures. Dosing may require renal adjustment and local protocol.
If hypotension persists after initial fluid resuscitation, start norepinephrine 0.05–0.1 mcg/kg/min IV infusion, titrating to a usual MAP target of at least 65 mm Hg; it restores vascular tone and organ perfusion.
Remove a suspected retained tampon, packing, or other foreign body immediately and obtain urgent surgical/obstetric consultation for a wound or deep source.
Definitive Care & Disposition
Admit patients with shock or organ dysfunction to the ICU; pursue urgent drainage, debridement, or other source control. Narrow antibiotics when cultures and the likely organism are known. If group A streptococcal infection is confirmed, use penicillin G 4 million units IV every 4 hours plus clindamycin 900 mg IV every 8 hours (check local protocol); involve infectious disease and surgery early. IVIG may be considered with specialists for severe streptococcal TSS, but evidence and dosing vary—check institutional guidance.
How This One Kills
Treating the rash and hypotension while leaving a tampon, infected wound, or necrotic tissue in place allows toxin production and shock to continue. Source control is part of resuscitation, not a later administrative task.
The Atypical Presentation
The classic diffuse rash may be faint, absent early, or obscured in darker skin; desquamation is delayed, and cultures can be negative in toxin-mediated staphylococcal disease. Older adults, postpartum patients, and partially treated patients may present chiefly with confusion, hypotension, or organ injury. Let shock plus a plausible source drive early treatment rather than waiting for a textbook rash or confirmatory culture.
Back to Our Patient
Back to 24-year-old Elena, who has a diffuse erythematous rash and persistent hypotension: recognize possible TSS and treat this as shock before waiting for confirmation. Resuscitate with reassessed crystalloid boluses, send cultures without delaying broad antibiotics, and start norepinephrine if hypotension persists. Examine for and remove a tampon or other foreign body, and urgently involve the team needed for source control. She requires ICU admission while the source is controlled and antimicrobials are narrowed to culture results.
Patient Presentation to Attending
“This is a 24-year-old with abrupt systemic illness, diffuse erythematous rash, and persistent hypotension after an initial liter of crystalloid. She is flushed and shivering, with impaired perfusion; the source is not yet established, and the rash has no reported mucosal erosions. I am concerned for toxic shock syndrome, including staphylococcal or streptococcal infection. I have obtained cultures without delaying vancomycin, clindamycin, and broad gram-negative/anaerobic coverage, and I am reassessing fluid response and starting norepinephrine if needed. I recommend immediate source inspection and removal or drainage as indicated, with ICU admission.”
Study Directive
From memory, list three likely TSS sources and the immediate source-control action for each.
Practice stating the empiric antibiotic combination and the reason clindamycin is included.
Review your sepsis pathway for fluid reassessment, vasopressor initiation, and ICU escalation.
Key Medications
Vancomycin: 20–25 mg/kg IV loading dose, maximum 3,000 mg; maintenance is individualized by renal function, exposure monitoring, and institutional protocol.
Clindamycin: 900 mg IV every 8 hours for toxin suppression.
Piperacillin–tazobactam: 4.5 g IV every 6–8 hours; adjust for renal function and local protocol.
Norepinephrine: start 0.05–0.1 mcg/kg/min IV and titrate to perfusion/MAP.
Confirmed group A streptococcal TSS: penicillin G 4 million units IV every 4 hours plus clindamycin 900 mg IV every 8 hours.
IVIG is not routine; dosing varies. Consult infectious disease and check institutional guidance.
High-Yield Pearls
Do not wait for late desquamation or a positive culture to act on shock with a plausible toxin-mediated source.
A retained foreign body or necrotic source can sustain illness despite appropriate antibiotics.
The Mimics
Septic shock without TSS — no toxin-associated syndrome is required; confusing them can lead to missed source control or failure to add toxin-suppressing therapy.
Kawasaki disease — typically a febrile mucocutaneous illness in children with a different time course; misclassification risks missing acute shock and urgent infection treatment.
Scarlet fever — pharyngitis with a sandpaper-like rash but generally without shock or acute multiorgan dysfunction; assuming TSS can prompt unnecessary ICU care, while the reverse error is more dangerous.
Drug reaction / severe cutaneous adverse reaction — medication exposure and characteristic mucosal or skin injury may distinguish it; mistaking infection for a drug reaction can delay antibiotics and source control.
Board Question
A patient with a diffuse erythematous rash and shock has suspected toxic shock syndrome. Which initial antimicrobial strategy best addresses both broad empiric coverage and toxin suppression? -
AVancomycin alone -
BClindamycin alone -
CVancomycin, clindamycin, and broad-spectrum source coverage -
DWait for cultures before antibiotics
Reveal answer
Correct: C
Empiric treatment should cover likely pathogens and include a protein-synthesis inhibitor such as clindamycin to suppress toxin production. Cultures are useful but must not delay antibiotics in shock.
4 of 6
Vasa Previa
In vasa previa, unprotected fetal vessels cross near the cervix and can rupture with membrane rupture or labor. The fetus may exsanguinate rapidly while the...
At 34 weeks, 28-year-old Priya feels a sudden warm gush as her membranes rupture. Blood appears on the sheet, but she is alert and her blood pressure is unchanged. On the monitor, the fetal heart rate drops and stays low. The operating team is being called. What cannot wait?
Before You Read
Why can a small amount of bleeding be catastrophic for the fetus but not the mother?
What should you do when bleeding follows membrane rupture and the fetal tracing deteriorates?
Which antenatal measures help—and which must never delay emergency delivery?
Why It Matters
In vasa previa, unprotected fetal vessels cross near the cervix and can rupture with membrane rupture or labor. The fetus may exsanguinate rapidly while the mother remains hemodynamically stable.
When to Think of It
Suspect vasa previa with painless vaginal bleeding after spontaneous or artificial membrane rupture, especially with fetal bradycardia, recurrent decelerations, or a sinusoidal tracing. Known risk factors include velamentous cord insertion, bilobed or succenturiate placenta, and low-lying placenta; antenatal ultrasound diagnosis changes the plan before labor begins.
Sick or Not Sick
The crucial call is whether there is bleeding with fetal compromise after membrane rupture. If so, treat as fetal hemorrhage and proceed to immediate cesarean delivery; maternal stability does not reassure you about fetal blood loss.
The First Fifteen Minutes
Activate obstetrics, anesthesia, neonatal, and operating-room teams. Begin continuous fetal monitoring, establish IV access, obtain type and crossmatch, and assess maternal stability—but do not delay delivery for confirmatory testing when fetal compromise is present.
Persistent fetal bradycardia or significant bleeding after membrane rupture: proceed to emergency cesarean delivery; delivery is definitive treatment. Do not wait for an Apt test or ultrasound in an unstable fetal situation.
If preterm delivery is anticipated and time permits without delaying urgent delivery, give betamethasone 12 mg IM; repeat once in 24 hours for fetal lung maturation when within the appropriate gestational-age window per obstetric protocol.
If delivery is expected before 32 weeks, consider magnesium sulfate 4–6 g IV loading dose over 20–30 minutes, then 1–2 g/hour IV for fetal neuroprotection per institutional protocol. Neither medication should delay emergency delivery.
Definitive Care & Disposition
Deliver by cesarean before labor or membrane rupture when vasa previa is diagnosed antenatally; timing is individualized by gestational age, symptoms, and local obstetric practice, often in the late-preterm period. At delivery, the neonatal team should be ready for immediate resuscitation and transfusion if fetal blood loss is suspected. A stable, prenatally diagnosed patient requires obstetric planning and delivery in a center with appropriate maternal and neonatal resources.
How This One Kills
Assuming the mother’s stable vital signs mean the bleeding is minor. In vasa previa, the blood may be fetal, and a small visible volume can represent life-threatening blood loss for the fetus.
The Atypical Presentation
Bleeding may be scant, intermittent, or initially absent, and fetal compromise may be the first unmistakable sign. Maternal vital signs can remain normal because the blood loss is fetal rather than maternal. In a patient with known risk factors—or any bleeding after membrane rupture—interpret the fetal tracing urgently and do not rely on visible blood volume to judge severity.
Back to Our Patient
Back to 28-year-old Priya: bleeding follows membrane rupture, and the fetal heart rate remains low despite her stable blood pressure. Recognize suspected vasa previa with fetal compromise, risk-stratify this as an immediate delivery emergency, and proceed to cesarean without waiting for confirmatory testing or antenatal medications. The neonatal team prepares to resuscitate and assess for fetal blood loss; Priya receives maternal evaluation and postpartum care after delivery.
Patient Presentation to Attending
“This is a 28-year-old at 34 weeks with sudden vaginal bleeding immediately after membrane rupture and persistent fetal bradycardia. She is alert and hemodynamically stable, but the fetal tracing is concerning; the timing raises concern for ruptured fetal vessels rather than maternal hemorrhage. I am treating this as suspected vasa previa with fetal compromise. I recommend immediate cesarean delivery with obstetric, anesthesia, and neonatal teams present; no confirmatory test or medication should delay delivery.”
Study Directive
Draw the anatomic difference between vasa previa and placenta previa from memory.
Practice an emergency handoff that explains why maternal stability does not reassure you about fetal status.
Review local guidance for planned delivery timing and antenatal corticosteroid eligibility in diagnosed vasa previa.
---
Key Medications
Betamethasone: 12 mg IM, repeat in 24 hours, when preterm delivery is anticipated and time permits; use the obstetric protocol for gestational-age eligibility.
Magnesium sulfate for fetal neuroprotection: 4–6 g IV over 20–30 minutes, then 1–2 g/hour when delivery before 32 weeks is expected; dosing protocols vary, so check institutional guidance.
These medications are not substitutes for delivery when fetal compromise is present. Pediatric resuscitation and transfusion are weight-based.
High-Yield Pearls
Vasa previa bleeding is potentially fetal blood loss: visible volume and maternal vital signs can be misleading.
A sinusoidal fetal tracing is ominous for fetal anemia and should accelerate—not postpone—definitive care.
The Mimics
Placenta previa — typically painless maternal bleeding with fetal vessels not exposed over the cervix; confusing it can obscure fetal exsanguination and delay urgent delivery.
Placental abruption — often painful bleeding with uterine tenderness or hypertonicity; mislabeling vasa previa as abruption can miss the need for immediate fetal-directed rescue.
Cervical or vaginal bleeding — a visible local lesion may explain bleeding, but fetal heart-rate deterioration after rupture still demands urgent evaluation; assuming a benign source risks missing fetal hemorrhage.
Board Question
At 34 weeks, a patient has vaginal bleeding immediately after rupture of membranes, followed by persistent fetal bradycardia. The mother is hemodynamically stable. What is the best next step? -
AWait for an Apt test to confirm fetal blood -
BObtain a formal ultrasound before deciding -
CProceed to emergency cesarean delivery -
DGive antenatal corticosteroids and reassess in 24 hours
Reveal answer
Correct: C
Bleeding after membrane rupture with persistent fetal bradycardia suggests ruptured fetal vessels and possible fetal exsanguination. Maternal stability and confirmatory testing should not delay emergency delivery.
Bleeding or fetal bradycardia after membrane rupture should raise concern for fetal-vessel hemorrhage and prompt immediate obstetric escalation for cesarean delivery.
Real-world screening and outcome data underscore the value of diagnosing vasa previa before labor so delivery can be planned before fetal hemorrhage occurs.
5 of 6 · Procedure Corner
Resuscitative Hysterotomy
Maternal cardiac arrest with a gravid uterus at or above the umbilicus (typically about 20 weeks or more), when initial resuscitation has not produced...
Also known asperimortem cesarean delivery · resuscitative cesarean delivery · perimortem cesarean section
0:00 / –:––AI‑generated audio
Indications
Maternal cardiac arrest with a gravid uterus at or above the umbilicus (typically about 20 weeks or more), when initial resuscitation has not produced return of spontaneous circulation (ROSC).
Begin preparation immediately when arrest is recognized; do not wait for fetal monitoring, confirmation of fetal viability, or transport to the operating room. Aim for delivery by 5 minutes after arrest when feasible, without delaying standard resuscitation.
Contraindications
In ongoing maternal cardiac arrest with a sufficiently enlarged uterus, there are no absolute contraindications; the procedure is performed primarily to improve maternal resuscitation, with potential fetal benefit.
If ROSC occurs before incision, reassess the need to proceed based on maternal status and obstetric indications rather than continuing automatically.
Equipment & Prep
Scalpel, scissors, clamps, gloves, suction if available, cord clamps or ties, and supplies for newborn ventilation and thermal support.
Assign roles for chest compressions, airway/ACLS, procedure, newborn care, and timekeeping. Continue high-quality CPR and standard ACLS, including indicated defibrillation.
Perform at the arrest location; use aseptic technique as feasible, but do not delay incision for sterile preparation or equipment retrieval.
Steps
Announce resuscitative hysterotomy, note the arrest time, and start the procedure as soon as the indication is recognized. Keep the patient supine on a firm surface.
Continue chest compressions and ACLS. Use manual left uterine displacement until uterine evacuation if it can be done without interrupting compressions.
Make a rapid vertical midline abdominal incision, extending through the abdominal wall to expose the uterus. Control the incision and extend it as needed for access.
Incise the uterus under direct vision, taking care to avoid fetal injury; extend the uterine incision sufficiently for delivery.
Deliver the fetus and clamp/cut the cord. Hand the newborn to the neonatal team while resuscitation continues.
Deliver the placenta if readily possible, then address uterine tone and bleeding. Continue maternal resuscitation and arrange definitive operative care and hemorrhage control.
Confirmation & Success Criteria
Confirm delivery and uterine decompression; assess for ROSC while continuing ACLS.
The operational goal is rapid delivery, ideally by 5 minutes after arrest. Do not delay or interrupt maternal resuscitation to meet a time target.
Assess the newborn promptly and provide neonatal resuscitation as indicated. Lack of immediate ROSC does not mean the procedure was inappropriate or unsuccessful.
Complications & Rescue
Hemorrhage, uterine or adjacent-organ injury, fetal laceration, and exposure-related injury are possible. Extend exposure under direct vision and control bleeding; obtain immediate obstetric and surgical support when available.
If access or delivery is difficult, improve exposure and extend the incision rather than applying force or delaying resuscitation.
Continue ACLS throughout; treat reversible causes of arrest and manage postpartum hemorrhage after delivery.
Aftercare & Documentation
Transfer for definitive operative management, hemorrhage control, and critical care as soon as feasible; continue maternal and neonatal resuscitation and monitoring.
Document arrest and incision times, gestational estimate or fundal height, resuscitation performed, delivery time and findings, maternal response, complications, and disposition.
Arrange team debriefing and appropriate support for the family and staff.
6 of 6 · Procedure Corner
Reduction of Nuchal Cord
A cord is found around the newborn’s neck after delivery of the head during vaginal birth.Reduce a loose cord if it can be moved gently without delaying...
Also known assomersault maneuver · nuchal cord management
0:00 / –:––AI‑generated audio
Indications
A cord is found around the newborn’s neck after delivery of the head during vaginal birth.
Reduce a loose cord if it can be moved gently without delaying delivery or placing traction on the cord.
Contraindications
Do not force a tight cord over the head or pull on it.
Avoid routine clamping and cutting before delivery of the shoulders when the infant can be delivered through the loop or by a somersault maneuver.
Equipment & Prep
Gloves and the usual supplies for vaginal birth and newborn care.
Ensure the birth team is ready to support the infant and proceed with delivery. No special instrument is required for gentle reduction.
Steps
After the head is born, support it and check for a cord around the neck.
If the cord is loose, gently slip it over the infant’s head.
If it will not pass easily, stop; do not pull or force it.
For a tight cord, keep the infant’s head close to the maternal thigh or perineum and guide the body through the loop using a somersault maneuver.
Once the body is delivered, unwind the cord if needed and assess the newborn.
Confirmation & Success Criteria
Confirm that the infant is fully delivered and the cord is no longer constricting the neck.
Assess newborn breathing, tone, and heart rate; begin neonatal resuscitation if indicated.
Complications & Rescue
Forceful manipulation can injure the infant or cord; stop if gentle reduction is unsuccessful and use the somersault maneuver when feasible.
If the cord is too tight to permit delivery by these methods, rapidly obtain experienced obstetric assistance. Clamping and cutting may be necessary when no safe alternative allows delivery; clamp twice and cut between clamps, then expedite birth and assess the newborn.
Avoid delaying delivery or applying traction to the cord. Treat neonatal compromise according to newborn resuscitation guidance.
Aftercare & Documentation
Continue routine maternal and newborn assessment, including evaluation for bleeding, cord injury, and neonatal transition.
Document the finding, maneuver used, any clamping or cutting, newborn condition, and interventions provided.
Yesterday’s Differential
The daily puzzle — from editions past
A quick test of recall from prior editions. Commit to an answer before you check.
From yesterday's edition
A thin, tremulous 34-year-old woman presents with palpitations, heat intolerance, and weight loss over two months. She is anxious and diaphoretic. What’s the diagnosis, and the first move?
Check your answer
Hyperthyroidism. Send TSH and T4 in any patient with unexplained sinus tachycardia or new AF, and control thyrotoxic atrial tachydysrhythmias with intravenous beta-blockade (titrated IV propranolol boluses or an esmolol infusion) while treating the underlying thyrotoxicosis; escalate to thyroid-storm management when rates exceed 200 bpm.
From the September 23 edition
Today, three days ago: Epinephrine. What’s the adult ED dose, and the contraindication you’d most regret missing?
Check your answer
Cardiac arrest: 1 mg IV/IO q3–5 min of 0.1 mg/mL solution. Anaphylaxis: 0.3–0.5 mg IM of 1 mg/mL into lateral thigh q5–15 min. Infusion: commonly 0.01–0.5 mcg/kg/min titrated to effect. No absolute contraindication in cardiac arrest or anaphylaxis.
From the September 16 edition
A 29-year-old develops profuse vomiting 10 hours after eating wild mushrooms, improves for 12 hours, then develops jaundice and INR elevation. Which intervention is most appropriate now?
ADischarge after symptom control
BImmediate high-dose corticosteroids
CPoison-center consultation, NAC, serial liver testing, and early transplant-center evaluation
DRoutine antibiotics for presumed bacterial gastroenteritis
Reveal answer
Correct · C
Amatoxin poisoning classically has delayed GI symptoms followed by a deceptive improvement and hepatotoxic phase. Early toxicology involvement, NAC, intensive monitoring, and transplant-center referral are critical.
Journal Watch
From the FOAMed wire
Notable posts and reviews from the last week, ranked by relevance to today’s lead and source trust.
Journal Feed covers syncope vs. presyncope and hospitalization, discharge of pediatric anaphylaxis, and autonomous oxygen titration. The post Journal Feed Weekly Wrap-Up appeared first on emDocs .
Podcast Picks
Two for the shift
Critical Care Perspectives in Emergency Medicine2026-09-01
Brain damage and death from ischemic encephalopathy is common after cardiac arrest. Animal studies and limited observational studies suggest that high oxygen exposure after ROSC may result in neuronal death. As such, it is hypothesized that limiting oxygen exposure (conservative oxygen) many attenuate reperfusion injury and improve outcomes. At present,...
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Critical Care Perspectives in Emergency Medicine
Published
2026-09-01
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Critical Care Perspectives in Emergency Medicine, Critical Care Perspectives in Emergency Medicine
Dr. Cho Espinosa—surgical intensivist, OB-GYN, and flight doc—joins me to overhaul how we handle catastrophic post-partum hemorrhage. Moving past standard ivory-tower guidelines, Sho dives deep into high-yield...
Annals Journal Watch
From the specialty flagship
One recent paper from Annals of Emergency Medicine worth knowing on shift, chosen for practice impact — independent of today’s topics.
Foster AA, Hoffmann JA, Addo N, et al. · PMID 42788929
This observational study asks which characteristics of ED patients with a positive youth suicide screen are associated with a subsequent attempt. It addresses a consequential disposition question residents face when a screening result alone cannot establish risk.
Bottom line → After a positive youth suicide screen, complete an individual risk assessment and safety plan rather than using the screen alone to decide disposition.
UMEM Pearl
Matched to today’s topics
A clinical pearl from the University of Maryland EM group’s Educational Pearls, tied to today’s differential.
Chest pain in pregnancy warrants a serious cardiac evaluation: cardiovascular disease is the leading cause of maternal death in the US.
Bottom Line: Move over, post-partum hemorrhage, because cardiovascular causes reign supreme! In a new study based on CDC data from 2005-2024, cardiovascular causes were the number one cause of death in pregnancy overall (well, except for COVID…).
Critical Care Corner
Matched to today’s topics
A critical-care reference from LITFL’s Critical Care Compendium, tied to today’s differential.
In toxic shock syndrome, toxin-driven capillary leak and vasodilation can rapidly turn fever and rash into shock requiring resuscitation and source control.
Staphylococcal Toxic Shock Syndrome: staphylococcal toxin producing infection -> intoxication of exotoxins (TSS-1, 2 or 3 enterotoxin)-> toxic shock syndrome
Pharmacology Corner
Two drugs for the shift
One antimicrobial and one other ED workhorse — selected daily, with sources and last-reviewed dates so every dose is cross-checkable.
Antimicrobial of the Day
Cefuroxime
Second-generation cephalosporin (oral and IV)
Indication
Community-acquired pneumonia, otitis media/sinusitis, SSTI, early Lyme disease, uncomplicated UTI, and surgical prophylaxis.
What’s your dose? — reveal dosing & cautions
ED Dose
PO: 250–500 mg q12h. IV: 750 mg–1.5 g q8h. Early Lyme: 500 mg PO q12h x14–21 days. Pediatric oral: 20–30 mg/kg/day divided q12h.
Renal Adjustment
Reduce IV frequency in CrCl < 20 mL/min; HD: dose after dialysis.
ED Pearl
One of the few oral cephalosporins with reliable activity in early Lyme disease — a useful alternative to doxycycline in pregnancy and young children.
Cardiac arrest special circumstances, severe hyperkalemia with unstable ECG, calcium channel blocker overdose, massive transfusion hypocalcemia, and severe hypocalcemia when central access is available.
What’s your dose? — reveal dosing & cautions
ED Dose
1 g IV of 10% calcium chloride, preferably through central line; may repeat based on ECG/hemodynamics/ionized calcium.
Renal Adjustment
No renal adjustment for emergency use; monitor calcium/phosphate with repeated doses.
Contraindications
Peripheral administration carries tissue necrosis risk with extravasation; caution in digoxin toxicity and hypercalcemia.
Interactions
Incompatible with bicarbonate in same line; ceftriaxone precipitation risk in neonates; digoxin arrhythmia concern.
Monitoring
ECG, ionized calcium, IV site/extravasation, hemodynamic response.
ED Pearl
Calcium chloride gives more elemental calcium than gluconate but is harsher on veins — use it when crashing/central access exists, not casually through a tenuous peripheral IV.
For educational use only. Verify dosing against the FDA label and your institution’s pharmacy resources before administering.
ECG of the Day
Channelopathy
Arrhythmogenic Right Ventricular Dysplasia (ARVD)
A young patient with exertional syncope, T-wave inversion in V1-V3 without a bundle branch block, and a small terminal notch after the QRS is carrying the second most common cause of sudden death in the young.
The Tracing
A 22-year-old soccer player collapses on the field, is briefly unconscious, and walks into your ED insisting he is fine. He mentions his cousin died suddenly in his twenties. Vitals are normal. His resting 12-lead shows deeply inverted T waves across V1, V2, and V3, yet the QRS is not broad in the way a bundle branch block would make it, and there is no RSR' pattern. Looking closely at the terminal QRS in V1 you can make out a small, low-amplitude deflection riding just after the S wave, before the ST segment. The precordial S waves rise sluggishly. On the monitor you catch a run of broad-complex beats with a left bundle pattern and an inferior axis.
T-wave inversion in the right precordial leads V1-V3 in the absence of RBBB (seen in ~85% of patients)
Epsilon wave — a small terminal deflection after the QRS, best seen in V1; the most specific finding, present in ~50%
Localized QRS widening in V1-V3 (>110 ms) and a prolonged S-wave upstroke (55 ms) in V1-V3
Ventricular ectopy of LBBB morphology, with frequent PVCs (>1000 per 24 hours)
Paroxysmal VT with LBBB morphology and an inferior axis (RVOT tachycardia)
Pearls
The epsilon wave is subtle and easily lost in baseline artifact. Recording at double speed (50 mm/s) and double amplitude, or using Fontaine bipolar precordial leads (RA on the manubrium, LA on the xiphoid, LL at V4), raises your yield for catching it.
The first presenting symptom can be sudden cardiac death, and there is usually a family history of it — take the exertional syncope and the family history as seriously as the tracing.
The broad-complex VT of ARVD arises from the diseased right ventricle, so it carries an LBBB morphology with an inferior axis — a useful fingerprint distinguishing RVOT tachycardia from other wide-complex rhythms.
Pitfalls
The right precordial T-wave inversion mimics benign persistent juvenile T-wave inversion, RBBB, and right-heart strain — but ARVD shows this pattern without a completed RBBB, which should prompt a second look.
A normal-looking resting ECG does not exclude ARVD; the diagnosis rests on a combination of clinical, ECG, and imaging (echo, cardiac MRI) features per the 2010 Task Force Criteria.
Reassuring and discharging a young syncope patient with these findings misses a high-risk substrate — syncope from cardiac arrest, unsuppressed arrhythmias, or a family history of arrest all mark high risk of sudden death.
At the Bedside
Do not clear this athlete. Admit for cardiology and electrophysiology evaluation with echo and cardiac MRI. Arrhythmia suppression is with beta-blockers, amiodarone, or sotalol; patients with any high-risk feature (syncope from cardiac arrest, recurrent unsuppressed arrhythmias, or first-degree family history of cardiac arrest) need urgent ICD implantation.
For educational use only. Verify ECG interpretation against the LITFL entry and your institution’s practice before clinical decision-making.
Case of the Day
From the lead · Shoulder Dystocia
Self-Examination
Test Your Understanding
After delivery of the fetal head, the shoulders do not deliver with gentle traction. Which is the most appropriate initial maneuver? -
AFundal pressure -
BMcRoberts positioning with suprapubic pressure -
CForceful downward traction on the head -
DImmediate cesarean delivery
Reveal answer
Correct answer · B
McRoberts positioning, commonly combined with suprapubic pressure, is the initial maneuver. Fundal pressure and forceful traction can worsen impaction and cause injury.
Study Pace4 topics today; Issue 74 of 94 — OB/GYN (Weeks 41 A-41 B)Deadline · June 1, 2026