An Emergency Medicine Broadsheet
·Phoenix·
Est. MMXXVI
Blue Fish Med · Today's Topic
Transgender Care in the ED, Part 1
Assumptions about anatomy, sexual activity, or hormone use can delay time-critical diagnoses. Respectful, specific questions improve both diagnostic accuracy and trust.
Also known astransgender health · gender-affirming care · gender dysphoria care
A 27-year-old transgender man sits forward on the stretcher, one hand pressed to his lower abdomen. He says the pain began as a dull ache and now catches sharply when he moves; he has also noticed a little spotting. He has not had a period in months and is unsure whether his hormone medication matters. The pregnancy test is pending, and the next question is yours to ask.
— What’s your move? Read on.
Before you read
When should pregnancy enter the differential despite testosterone use or amenorrhea?
How do you make the evaluation affirming without missing an emergency?
When to Think of It
Use the patient’s stated name and pronouns, and ask what terms they prefer for body parts and care. Clarify relevant organs, surgeries, hormone use, and pregnancy possibility rather than inferring them from gender identity or appearance. Consider pregnancy in any patient with a uterus and ovaries who could have had sperm exposure—even with testosterone use, amenorrhea, or contraception.
Sick or Not Sick
The key fork is physiologic instability or a time-critical condition versus a stable patient suitable for a focused workup. In pelvic pain or bleeding, do not let gender identity or testosterone use rule out pregnancy; identify shock, peritonitis, or concerning ultrasound findings early.
The First Fifteen Minutes
Ask permission before sensitive questions or examination; explain why each question matters, offer a chaperone, and protect privacy.
For a patient with pregnancy potential and abdominal/pelvic pain or bleeding → obtain a urine or serum pregnancy test; pregnancy testing is based on organs and exposure, not gender identity.
For significant pain in a stable patient → acetaminophen 1,000 mg PO or IV; it provides analgesia without obscuring the need to investigate the cause. Do not exceed 4,000 mg/day in a healthy adult; use a lower limit with liver disease, heavy alcohol use, or other risk factors.
For instability or suspected internal bleeding → establish large-bore IV access, send type and crossmatch, and activate the hemorrhage/transfusion pathway as indicated; resuscitation and urgent specialty involvement take priority over a complete history.
Definitive Care & Disposition
Evaluate the presenting complaint using the relevant anatomy and risks: for pelvic pain, that may include pregnancy testing, pelvic ultrasound, and gynecology consultation. Do not require a genital examination unless it is clinically necessary, and obtain consent before proceeding. Continue or pause established hormone therapy only after considering the acute condition and, when useful, consulting the patient’s prescriber; the ED visit is not a reason to discontinue it reflexively. Disposition depends on the underlying diagnosis, stability, and ability to complete urgent follow-up.
How This One Kills
The dangerous error is excluding pregnancy because the patient uses testosterone or has no periods—delaying diagnosis of ectopic pregnancy until rupture and hemorrhagic shock.
The Differential — What Else Looks Like This
Testosterone-associated amenorrhea — does not establish infertility; mistaking it for pregnancy exclusion can miss ectopic pregnancy.
Gender dysphoria or anxiety — may affect how a patient describes symptoms, but does not explain away focal tenderness or abnormal vital signs; anchoring can delay diagnosis.
Urinary infection — urinary symptoms or pyuria may coexist with another cause of pelvic pain; treating a urine result alone can miss a surgical emergency.
The Second-Day Story
A patient may be reluctant to disclose anatomy, sexual exposure, or hormone use after prior disrespectful care, and testosterone may make menstrual history unhelpful. Ask permission, explain the clinical reason for each question, and use neutral, specific language; when pregnancy is possible, test rather than relying on reported bleeding patterns or presumed fertility.
Back to Our Patient
Back to our 27-year-old patient. Recognize that lower abdominal pain and spotting merit a pregnancy test despite testosterone use and months without periods; respectfully ask about relevant anatomy and sperm exposure. He is initially stable, so risk-stratify with serial vitals, pregnancy testing, and pelvic ultrasound rather than assuming a hormone-related cause. The test is positive, and ultrasound shows an adnexal mass with free fluid; gynecology is consulted urgently, and he is admitted for definitive management of a suspected ectopic pregnancy.
Patient Presentation to Attending
How you’d present this patient on the floor — tight, pertinent positives and negatives, no rambling
“This is a 27-year-old transgender man presenting with worsening lower abdominal pain and new spotting. The pain started as a dull ache and is now sharp with movement; he reports months of amenorrhea while taking testosterone. He is sexually active with a partner who produces sperm and has not had sterilization. He is currently hemodynamically stable, with focal lower abdominal tenderness but no generalized peritonitis. His pregnancy test is positive, and pelvic ultrasound shows an adnexal mass with free fluid. I’m concerned for ectopic pregnancy; I’ve involved gynecology urgently and am arranging admission and ongoing assessment for bleeding.”
Study Directive
Practice a 30-second, permission-based sexual and reproductive history using neutral language. Review which organs and surgeries change the differential for pelvic pain, pregnancy, and routine screening; then write a one-sentence explanation for when you would offer a pregnancy test.
More in Today's Issue
6 additional topics
2 of 7
Transgender Care in the ED, Part 2
Hormone use and gender-affirming surgery can affect risk and anatomy, but neither should become an anchoring shortcut. Missing venous thromboembolism or a...
Also known astransgender health · gender-affirming care · gender dysphoria care
A 34-year-old transgender woman pauses mid-sentence to catch her breath, then points to her right calf: it has felt tight since yesterday. She takes estrogen and returned from a long flight three days ago. Her oxygen saturation is 91%, and the room seems suddenly quieter as the monitor beeps. The next move is not yet clear.
Before You Read
Which features change your threshold for evaluating a possible clot?
When is it reasonable to anticoagulate before imaging?
What should you do with the patient’s established hormone regimen during an acute event?
Why It Matters
Hormone use and gender-affirming surgery can affect risk and anatomy, but neither should become an anchoring shortcut. Missing venous thromboembolism or a postoperative complication can be fatal.
When to Think of It
Consider venous thromboembolism in a patient with new unilateral leg swelling, pleuritic pain, unexplained tachycardia, hypoxemia, or syncope. Ask about the specific hormone, route, dose, recent changes, personal clot history, smoking, immobility, and surgery. For postoperative symptoms, clarify the procedure and date; complications depend on the operation performed.
Sick or Not Sick
The critical call is whether the patient is unstable or has a high likelihood of a time-critical complication. Shock, severe hypoxemia, syncope, or rapidly progressive symptoms prompt resuscitation and urgent escalation; stable patients proceed through appropriate diagnostic testing. Do not attribute new symptoms to hormones or dysphoria before evaluating dangerous causes.
The First Fifteen Minutes
Obtain vital signs, ECG, focused cardiopulmonary and limb examination, and IV access; establish what hormones and surgeries the patient has had without delaying stabilization.
For suspected PE, choose imaging based on clinical probability and local protocol; pregnancy is not assumed from gender identity, and indicated imaging should not be delayed solely because radiation is a concern.
For likely acute VTE when imaging will be delayed and bleeding risk is acceptable → enoxaparin 1 mg/kg SC every 12 hours; it provides therapeutic anticoagulation while testing proceeds. Adjust for renal impairment and individual circumstances.
For hemodynamic instability, imminent procedure, or a need for rapidly reversible anticoagulation → unfractionated heparin, commonly 80 units/kg IV bolus followed by 18 units/kg/hour IV infusion for VTE; titrate to the institutional monitoring protocol. Dosing protocols vary—check your institutional reference.
Do not stop or change established hormones automatically; weigh the acute risk, patient preference, and specialist input against the consequences of an abrupt change.
Definitive Care & Disposition
Confirm or exclude VTE with appropriate imaging and evaluate for other causes of cardiopulmonary symptoms. Patients with confirmed PE require disposition based on hemodynamics, oxygen needs, RV strain, bleeding risk, and ability to complete outpatient treatment; instability or significant cardiopulmonary compromise warrants admission and specialist escalation. For a suspected surgical-site complication, involve the relevant surgical team early and assess for infection, bleeding, obstruction, or wound breakdown. Discuss hormone route and ongoing therapy with the patient and their prescriber or specialist rather than making a blanket long-term change in the ED.
How This One Kills
The dangerous failure is dismissing hypoxemia, tachycardia, or unilateral swelling as “just hormones” or anxiety, delaying diagnosis of PE until cardiovascular collapse.
The Atypical Presentation
Patients may not know the name or dose of a hormone, may use nonprescribed formulations, or may avoid discussing surgery after prior stigmatizing care. Ask specific, nonjudgmental questions and verify medications when possible; older patients or those with gradual symptoms may have little classic calf pain or chest pain, so unexplained hypoxemia or tachycardia still warrants a serious evaluation.
Back to Our Patient
Back to our 34-year-old patient with calf tightness, recent long-haul travel, and oxygen saturation of 91%. Recognize possible VTE and assess stability immediately; she is tachycardic but maintaining blood pressure, so proceed with urgent PE evaluation while establishing access and monitoring. Because imaging is not immediately available, clinical suspicion is high, and there is no evident bleeding contraindication, begin therapeutic anticoagulation while arranging imaging. Imaging confirms PE; she is admitted for ongoing treatment and risk assessment, with her hormone plan reviewed in consultation rather than changed reflexively.
Patient Presentation to Attending
“This is a 34-year-old transgender woman with new right calf tightness and shortness of breath after a long flight three days ago. She takes estrogen; the route and dose are being verified. She is tachycardic and hypoxemic at 91% but maintaining her blood pressure, with unilateral calf swelling and no evident active bleeding. Her ECG shows sinus tachycardia, and her presentation is concerning for acute VTE with PE. Imaging is being arranged urgently; given the delay, high clinical suspicion, and no apparent contraindication, I’ve started therapeutic anticoagulation. I recommend admission for confirmed PE and specialist review of her ongoing hormone regimen.”
Study Directive
Practice a focused hormone and surgery history in under one minute. Then review your local PE diagnostic and anticoagulation pathways, including which findings require admission and how your heparin infusion is monitored.
Key Medications
Enoxaparin: 1 mg/kg SC every 12 hours for therapeutic VTE treatment; adjust for renal function and clinical context.
Unfractionated heparin: commonly 80 units/kg IV bolus, then 18 units/kg/hour IV infusion for VTE; use the local titration protocol. Weight caps and monitoring targets vary—check institutional guidance.
Gender-affirming hormone regimens vary by medication, formulation, and patient; do not use a generic ED initiation or adjustment dose. Verify the actual regimen and consult a current drug reference or relevant specialist when needed.
High-Yield Pearls
Hormone route and formulation matter to risk assessment; “on hormones” is not a sufficiently specific medication history.
Postoperative emergencies are procedure-specific: clarify what operation was performed rather than assuming anatomy or complications.
The Mimics
Cellulitis — warmth, erythema, and skin tenderness may dominate, but unilateral swelling does not exclude DVT; confusing them can delay anticoagulation.
Musculoskeletal calf strain — pain after activity without systemic findings may fit, but it does not account for unexplained hypoxemia or dyspnea; anchoring risks missing PE.
Panic attack — may cause breathlessness and tachycardia, but does not explain objective hypoxemia; prematurely labeling symptoms as anxiety can be catastrophic.
Board Question
A hemodynamically stable transgender woman taking estrogen presents with unilateral leg swelling and new hypoxemia. Imaging will be delayed, clinical suspicion for PE is high, and there is no apparent bleeding contraindication. What is the best next step?
AAttribute symptoms to anxiety and discharge with follow-up.
BStop estrogen permanently and defer further evaluation.
CBegin therapeutic anticoagulation while arranging urgent diagnostic imaging.
DWait for imaging before taking any action in all cases.
Reveal answer
Correct: C
When PE is strongly suspected, imaging is delayed, and bleeding risk is acceptable, empiric therapeutic anticoagulation is appropriate while evaluation proceeds. Hormone use informs the history but does not replace diagnostic assessment or justify an automatic permanent medication change.
3 of 7
Breech Presentation
Breech labor can become an emergency when the cord is compressed or the head is trapped after the body delivers. Avoiding delay and avoiding untrained...
Also known asbreech lie · footling breech · frank breech · complete breech
A 29-year-old at 39 weeks grips the side rail as another contraction tightens across her abdomen. The nurse calls you to the room: a small foot is visible, and the fetal heart rate on the monitor has slowed. The delivery team is on its way, but the next contraction is already building.
Before You Read
What single finding determines whether you prepare for urgent cesarean delivery or an imminent vaginal birth?
What should you do—and avoid doing—if the breech is delivering?
What is the immediate response to a prolapsed umbilical cord?
Why It Matters
Breech labor can become an emergency when the cord is compressed or the head is trapped after the body delivers. Avoiding delay and avoiding untrained traction can protect both patient and newborn.
When to Think of It
Breech presentation is suspected when the presenting part is the buttocks or feet rather than the head; confirm with bedside ultrasound if it will not delay care. A foot at the introitus, visible buttocks, or a palpable presenting part with strong contractions may signal imminent birth.
Sick or Not Sick
The call that matters most is imminent vaginal delivery versus birth not imminent. If birth is not imminent, mobilize obstetrics and prepare for urgent cesarean delivery as indicated. If birth is unavoidable or already underway, support a controlled delivery with the most experienced available clinician.
The First Fifteen Minutes
Call obstetrics, anesthesia, neonatal resuscitation, and additional experienced help; note gestational age, presentation, fetal heart rate, and contraction pattern.
Position the patient with left lateral uterine displacement if supine; establish IV access and continuous fetal monitoring if feasible without delaying delivery.
If an umbilical cord prolapses → elevate the presenting part manually off the cord, position knee-chest or steep lateral with hips elevated, and expedite delivery; do not push the cord back into the uterus.
If the breech is delivering → do not pull on the fetus; support the body and follow the obstetric team’s maneuvers. Avoid delaying an imminent birth to transfer the patient.
After birth, if uterotonic prophylaxis is indicated and there is no contraindication → oxytocin 10 units IM once, or IV infusion per local protocol; it reduces postpartum hemorrhage by promoting uterine contraction.
Definitive Care & Disposition
If delivery is not imminent, urgent obstetric-led delivery planning—often cesarean delivery—is required; do not attempt external cephalic version in the ED. If vaginal breech birth is unavoidable, an experienced obstetric clinician should direct maneuvers and the neonatal team should assess the newborn promptly. Admit for delivery and postpartum monitoring; evaluate and treat hemorrhage, trauma, or neonatal compromise as indicated.
How This One Kills
The failure to fear is not simply “breech birth”—it is allowing the body to deliver while the after-coming head remains trapped, or leaving a prolapsed cord compressed, causing fetal hypoxia.
The Atypical Presentation
Breech may not be known before labor, especially with preterm birth, ruptured membranes, or limited prenatal care. A foot or buttocks may be mistaken for another structure, and fetal compromise can develop before a full examination is completed; use bedside ultrasound if immediately available, call obstetrics early, and let urgency—not diagnostic perfection—guide the response.
Back to Our Patient
Back to our 29-year-old at 39 weeks, with a foot visible and fetal heart rate slowing. Recognize breech presentation with fetal compromise and risk-stratify by the decisive finding: birth is imminent, so there is no time to delay for transfer to an operating room before preparing for delivery. Call obstetrics, anesthesia, and neonatal resuscitation, relieve cord compression if present, and support the birth without traction under the most experienced clinician’s direction. After delivery, give indicated oxytocin prophylaxis and admit the patient and newborn for obstetric and neonatal monitoring.
Patient Presentation to Attending
“This is a 29-year-old at 39 weeks in active labor with a foot visible at the introitus and fetal bradycardia on the monitor. She is contracting frequently, and the breech birth appears imminent. I have called obstetrics, anesthesia, and the neonatal team and am preparing for immediate delivery rather than delaying for transfer. I’m checking for cord prolapse and will relieve pressure on the cord if present. I will not apply traction to the fetus; I’ll support delivery under obstetric direction. After birth, we’ll assess maternal bleeding and neonatal status and arrange admission for both.”
Study Directive
Sketch a one-minute breech birth response from memory: team activation, imminent-versus-not decision, cord prolapse maneuver, and the “no traction” rule. Review local obstetric and neonatal escalation pathways with your department.
Key Medications
Oxytocin: 10 units IM once after delivery for postpartum hemorrhage prophylaxis, or IV infusion per institutional protocol. Avoid an IV bolus because of potential hemodynamic effects.
No medication reverses cord compression or corrects breech presentation; delivery is the definitive response. Pediatric medication and resuscitation doses are weight-based and should follow neonatal protocols.
High-Yield Pearls
With breech labor, the presenting part and whether birth is imminent—not the label alone—drive the immediate plan.
Never use traction to hasten delivery; cord compression and entrapment of the after-coming head are the disasters to prevent.
The Mimics
Cephalic presentation with a hand or foot alongside the head — ultrasound or careful obstetric assessment identifies the head as presenting; mistaking a compound presentation for breech can lead to the wrong delivery plan.
Cord prolapse without visible fetal parts — a cord may be palpable or seen after membrane rupture; missing it permits continued compression and fetal hypoxia.
Nonreassuring fetal heart rate from another cause — persistent bradycardia can have multiple causes; assuming it is only a transient contraction effect may delay urgent delivery.
Board Question
A patient in active labor has a prolapsed umbilical cord and persistent fetal bradycardia. What is the most appropriate immediate action while preparing for expedited delivery?
APush the cord back into the uterus.
BElevate the presenting part off the cord and position the patient to reduce compression.
CApply fundal pressure to hasten delivery.
DWait for ultrasound confirmation before intervening.
Reveal answer
Correct: B
Manually lifting the presenting part off the cord and positioning the patient to reduce compression are immediate temporizing measures while delivery is expedited. Do not push the cord back or delay for imaging.
For term breech birth, understand the maternal and neonatal trade-offs of planned vaginal delivery versus cesarean section when counseling and coordinating urgent obstetric care.
Pregnancy does not protect against acute coronary syndrome, pulmonary embolism, or aortic dissection, and some cardiovascular emergencies occur more often...
A 32-year-old at 30 weeks pauses on the walk from the waiting room, pressing a palm to the center of her chest. She says the pain began while carrying laundry upstairs and now reaches between her shoulder blades; the fetal monitor ticks steadily in the next room. Her blood pressure is 168/104 mm Hg, and the ECG is being printed.
Before You Read
Which dangerous causes must stay on the table beyond reflux and musculoskeletal pain?
Which test or treatment should not be withheld simply because the patient is pregnant?
What findings change the urgency and destination of care?
Why It Matters
Pregnancy does not protect against acute coronary syndrome, pulmonary embolism, or aortic dissection, and some cardiovascular emergencies occur more often during pregnancy or postpartum. Delaying maternal diagnosis can threaten both patient and fetus.
When to Think of It
Treat new chest pain, dyspnea, syncope, hypoxemia, ischemic ECG changes, or severe hypertension as potentially serious. Consider acute coronary syndrome, PE, aortic dissection, pneumothorax, and peripartum cardiomyopathy alongside common causes. Ask gestational age, postpartum status, vascular risk, thromboembolic risks, and whether pain is abrupt, exertional, pleuritic, or radiating.
Sick or Not Sick
The key call is whether there is maternal instability or a high-risk cause requiring immediate resuscitation and specialty intervention. Abnormal vital signs, syncope, persistent pain, ischemic ECG, neurologic or pulse deficit, or signs of heart failure demand urgent escalation; pregnancy does not justify delaying indicated imaging.
The First Fifteen Minutes
Assess airway, breathing, circulation, bilateral arm blood pressures when dissection is possible, ECG, cardiac monitoring, IV access, and focused maternal and fetal assessment. Obtain troponin and targeted labs; a normal initial ECG does not exclude ACS.
For suspected ACS without aspirin allergy or active major bleeding → aspirin 162–325 mg PO, chewed; it inhibits platelet aggregation and reduces thrombotic complications.
For ongoing ischemic chest pain with adequate blood pressure and no suspected right-ventricular infarction or recent phosphodiesterase-5 inhibitor use → nitroglycerin 0.4 mg SL every 5 minutes as needed, up to 3 doses; it reduces myocardial oxygen demand by decreasing preload.
For confirmed or strongly suspected ACS requiring anticoagulation → unfractionated heparin per the ACS protocol (commonly 60 units/kg IV bolus, maximum 4,000 units, then 12 units/kg/hour IV, maximum initial rate 1,000 units/hour); it limits further thrombus formation. Protocols vary—check local dosing and monitoring guidance.
For suspected PE with high clinical probability, delayed imaging, and no important bleeding contraindication → enoxaparin 1 mg/kg SC every 12 hours; it provides therapeutic anticoagulation. Adjust for renal function and clinical circumstances.
For suspected aortic dissection with severe hypertension → urgent specialist involvement and controlled IV beta-blockade; for example, labetalol 10–20 mg IV initially, then 20–80 mg IV every 10 minutes as needed to a maximum cumulative 300 mg. It reduces shear stress by lowering heart rate and blood pressure; dosing and targets require protocol-guided care.
Definitive Care & Disposition
Choose imaging to answer the suspected diagnosis; use CT angiography or other necessary imaging when indicated rather than delaying for pregnancy alone. Confirmed ACS, PE, dissection, heart failure, or persistent unexplained symptoms require specialty consultation and usually admission; instability requires critical care. Involve obstetrics early for maternal-fetal monitoring and delivery planning, but maternal stabilization comes first. Add antiplatelet therapy, revascularization, anticoagulation, blood-pressure control, or delivery planning according to the definitive diagnosis and specialists’ recommendations.
How This One Kills
The failure mode is labeling chest pain as normal pregnancy discomfort, reflux, or anxiety before excluding a time-critical maternal diagnosis—especially when abnormal vital signs or a concerning history are present.
The Atypical Presentation
Pregnancy can make baseline breathlessness, tachycardia, and reflux common, blurring early warning signs; serious disease may first appear as fatigue, nausea, or unexplained exertional limitation. Older patients, those with diabetes, and postpartum patients may lack classic crushing pain, so attend to vital signs, ECG, risk factors, and the trajectory of symptoms rather than relying on textbook pain description.
Back to Our Patient
Back to our 32-year-old at 30 weeks with exertional chest pain radiating to her back and blood pressure of 168/104. Recognize possible ACS or aortic dissection rather than assuming reflux; risk-stratify her as high risk because of severe hypertension and radiating pain. Obtain ECG and cardiac testing, monitor mother and fetus, and involve obstetrics and the appropriate specialty urgently. Her ECG shows ischemic changes and troponin is elevated; after assessing for contraindications, give aspirin and initiate ACS-directed care with protocol-guided anticoagulation. She is admitted for urgent cardiology-led evaluation and maternal-fetal monitoring.
Patient Presentation to Attending
“This is a 32-year-old at 30 weeks’ gestation with exertional central chest pain radiating between her shoulder blades. Her blood pressure is 168/104, and she has persistent pain without syncope or a focal neurologic deficit. The ECG shows ischemic changes, and her troponin is elevated. I’m concerned for acute coronary syndrome, while keeping aortic dissection in the differential given the radiation and severe hypertension. I’ve started monitoring, obtained IV access, involved obstetrics and cardiology, and am initiating ACS treatment after checking contraindications. She needs urgent admission for definitive cardiac evaluation and maternal-fetal monitoring.”
Study Directive
Draw a chest-pain-in-pregnancy differential from memory, then rehearse how vital signs, ECG, and symptom pattern direct the first diagnostic branch. Review your local pregnancy imaging pathway and ACS anticoagulation protocol.
Key Medications
Aspirin: 162–325 mg PO, chewed, for suspected ACS when not contraindicated.
Nitroglycerin: 0.4 mg SL every 5 minutes, up to 3 doses, for ongoing ischemic pain when blood pressure is adequate and contraindications are absent.
Unfractionated heparin for ACS: commonly 60 units/kg IV bolus (maximum 4,000 units), then 12 units/kg/hour IV (maximum initial rate 1,000 units/hour); use the local ACS protocol.
Enoxaparin for PE: 1 mg/kg SC every 12 hours; adjust for renal impairment and clinical context.
Labetalol: 10–20 mg IV initially, then 20–80 mg IV every 10 minutes as needed, maximum cumulative dose 300 mg; use protocol-guided blood-pressure targets and specialist input. Pregnancy and emergency dosing protocols vary—check an institutional reference if uncertain.
High-Yield Pearls
A normal initial ECG or troponin does not rule out early ACS; reassess persistent or evolving symptoms.
Pregnancy-related physiology can blur PE and heart-failure signals—hypoxemia or syncope is not routine pregnancy dyspnea.
The Mimics
Gastroesophageal reflux — burning after meals or when supine may support it, but does not explain ischemic ECG changes or hemodynamic instability; mislabeling ACS can delay reperfusion.
Musculoskeletal chest pain — reproducible tenderness supports a chest-wall cause, but does not exclude PE or ACS; anchoring on tenderness can be dangerous.
Normal pregnancy-related dyspnea — gradual breathlessness without hypoxemia or focal findings may be physiologic; new hypoxemia, syncope, or acute chest pain warrants a search for disease.
Board Question
A patient at 28 weeks’ gestation presents with acute chest pain, hypoxemia, and high clinical suspicion for PE. CT pulmonary angiography is indicated. What is the best approach?
ADefer imaging until after delivery because of fetal radiation exposure.
BPerform indicated diagnostic imaging without delaying solely because the patient is pregnant.
CUse a normal ECG to exclude PE.
DDiagnose PE from pregnancy-associated D-dimer elevation alone.
Reveal answer
Correct: B
Maternal diagnosis and stabilization are priorities; necessary imaging should not be withheld solely because of pregnancy. D-dimer and ECG alone cannot establish or exclude PE in this setting.
Use a pregnancy-specific approach to recognize and manage dangerous cardiac causes of chest pain, including acute coronary syndrome and aortic dissection.
For pregnant patients with chest pain and suspected pulmonary embolism, the pregnancy-adapted YEARS pathway helps guide D-dimer testing and decisions about chest imaging.
Breech birth is imminent and transfer for cesarean delivery is not feasible without delaying birth. If birth is not imminent, urgently involve obstetrics...
Also known asbreech birth · breech presentation
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Indications
Breech birth is imminent and transfer for cesarean delivery is not feasible without delaying birth. If birth is not imminent, urgently involve obstetrics and pursue the safest available delivery plan.
Steps
Call obstetrics, anesthesia, and neonatal resuscitation help; prepare maternal and newborn resuscitation equipment.
Allow spontaneous descent and delivery of the trunk when possible. Support the fetus without pulling on the legs, abdomen, or neck.
If the legs remain extended, a trained clinician may use the Pinard maneuver to flex and deliver them. If the arms are extended or nuchal, use a recognized arm-delivery maneuver such as Lovset’s.
For the aftercoming head, maintain flexion with a recognized maneuver such as Mauriceau–Smellie–Veit; an experienced clinician may use Piper forceps. An assistant may apply suprapubic pressure.
Confirmation & Success Criteria
The head and body deliver without excessive traction; assess and support newborn transition promptly, and assess the birthing patient for bleeding and trauma.
Complications & Rescue
Cord prolapse or compression, head entrapment, fetal hypoxia, and birth trauma may occur. If descent or delivery stalls, avoid forceful traction; summon senior obstetric help and prepare for the appropriate rescue, including operative delivery when feasible.
6 of 7 · Procedure Corner
Precipitous Delivery In the ER
Birth is imminent in the ED, with crowning or rapid progression that makes transfer unsafe or impractical.
Also known asprecipitous labor · rapid labor · unplanned out-of-hospital birth
0:00 / –:––AI‑generated audio
Indications
Birth is imminent in the ED, with crowning or rapid progression that makes transfer unsafe or impractical.
Steps
Call obstetrics, anesthesia, and neonatal resuscitation help; position the patient safely and prepare a warm resuscitation area.
Support the emerging head and perineum; allow controlled birth without pulling on the head. Check for a nuchal cord after the head delivers and manage it if present.
Allow restitution and delivery of the shoulders with gentle, controlled axial guidance. If the shoulders do not deliver readily, stop traction and begin the shoulder dystocia sequence.
Support the newborn’s body as it delivers; dry, warm, assess breathing and tone, and initiate neonatal resuscitation as indicated.
Confirmation & Success Criteria
Newborn is delivered and receives appropriate immediate assessment and support; maternal bleeding and genital-tract trauma are assessed.
Complications & Rescue
Shoulder dystocia, neonatal compromise, maternal laceration, and postpartum hemorrhage are key risks. For shoulder dystocia, call for help and use the established maneuver sequence; for neonatal compromise or maternal hemorrhage, initiate resuscitation and urgent specialist management.
7 of 7 · Procedure Corner
Shoulder Dystocia
After delivery of the head, the shoulders do not deliver with gentle axial traction and routine maternal effort.
Also known asimpacted shoulder · fetal shoulder dystocia
After delivery of the head, the shoulders do not deliver with gentle axial traction and routine maternal effort.
Steps
Announce shoulder dystocia, call for obstetric and neonatal help, note the time, and stop forceful traction. Do not apply fundal pressure.
Use McRoberts positioning and suprapubic pressure, directing pressure to dislodge the anterior shoulder.
If unresolved, proceed to an internal rotational maneuver or deliver the posterior arm, according to clinician training and the situation.
If needed, reposition to hands-and-knees if feasible and continue the shoulder dystocia algorithm with senior obstetric support.
Confirmation & Success Criteria
Both shoulders and the body deliver; document the head-to-body interval and maneuvers used, and assess the newborn and birthing patient for injury.
Complications & Rescue
Potential harms include neonatal hypoxia or brachial plexus injury and maternal hemorrhage or genital-tract trauma. If initial maneuvers fail, escalate promptly to experienced obstetric help and advanced rescue options; avoid excessive traction and fundal pressure.
Yesterday’s Differential
The daily puzzle — from editions past
A quick test of recall from prior editions. Commit to an answer before you check.
From yesterday's edition
A 58-year-old man presents with weeks of worsening dyspnea, orthopnea and ankle swelling. He is tachypneic with crackles at both bases. What’s the diagnosis, and the first move?
Check your answer
Dilated Cardiomyopathy. Use the ECG to raise suspicion, then confirm with echocardiography (ejection fraction < 40%, chamber dilatation) — recognition drives heart-failure therapy, telemetry for the malignant ventricular arrhythmias that cause sudden death, and referral for advanced management up to transplant evaluation in end-stage disease. Treat decompensated biventricular failure and don't be falsely reassured by a 'pseudoinfarct' pattern.
From the September 22 edition
Today, three days ago: Dobutamine. What’s the adult ED dose, and the contraindication you’d most regret missing?
Check your answer
2–20 mcg/kg/min IV infusion, titrated to perfusion/cardiac output; often requires concurrent vasopressor if hypotensive. Idiopathic hypertrophic subaortic stenosis/HOCM physiology, uncontrolled tachyarrhythmias.
From the September 15 edition
A 48-year-old woman has progressive difficulty seeing objects to either side while reading. Visual acuity is 20/20 in each eye, and confrontation testing shows loss of both temporal fields. Which structure is most likely compressed?
ARight optic tract
BOptic chiasm
CLeft optic nerve
DRight occipital cortex
Reveal answer
Correct · B
Optic chiasm. Crossing nasal retinal fibers at the chiasm carry temporal visual fields, so chiasmal compression produces bitemporal hemianopia. Optic tract and occipital lesions produce contralateral homonymous defects.
Journal Watch
From the FOAMed wire
Notable posts and reviews from the last week, ranked by relevance to today’s lead and source trust.
Brain damage and death from ischemic encephalopathy is common after cardiac arrest. Animal studies and limited observational studies suggest that high oxygen exposure after ROSC may result in neuronal death. As such, it is hypothesized that limiting oxygen exposure (conservative oxygen) many attenuate reperfusion injury and improve outcomes. At present,...
Source
Critical Care Perspectives in Emergency Medicine
Published
2026-09-01
Host
Critical Care Perspectives in Emergency Medicine, Critical Care Perspectives in Emergency Medicine
SCCM recently published updated guidelines on neuromuscular blockade in ARDS. The authors present five recommendations, the first being the most potentially influential on practice. “Paralyze patients with ARDS...
UMEM Pearl
Matched to today’s topics
A clinical pearl from the University of Maryland EM group’s Educational Pearls, tied to today’s differential.
For chest pain in pregnancy, cardiovascular disease’s leading role in maternal mortality is a reminder not to dismiss cardiac symptoms as normal gestational physiology.
Bottom Line: Move over, post-partum hemorrhage, because cardiovascular causes reign supreme! In a new study based on CDC data from 2005-2024, cardiovascular causes were the number one cause of death in pregnancy overall (well, except for COVID…).
Critical Care Corner
Matched to today’s topics
A critical-care reference from LITFL’s Critical Care Compendium, tied to today’s differential.
Chest pain in pregnancy can signal a cardiopulmonary emergency; maternal respiratory failure or haemodynamic instability makes pregnancy-specific intensive care immediately relevant.
Pregnancy and Intensive Care: Common reasons for admission include hypertensive disorders; haemorrhage; respiratory failure; sepsis; trauma
Pharmacology Corner
Two drugs for the shift
One antimicrobial and one other ED workhorse — selected daily, with sources and last-reviewed dates so every dose is cross-checkable.
Antimicrobial of the Day
Ceftazidime
Third-generation IV cephalosporin (anti-pseudomonal)
1–2 g IV q8h. Febrile neutropenia: 2 g IV q8h. Pediatric: 30–50 mg/kg/dose q8h (max 6 g/day).
Renal Adjustment
Dose-adjust when CrCl < 50 mL/min; HD: dose after dialysis.
Contraindications
Cephalosporin hypersensitivity.
Interactions
Minimal.
Monitoring
Renal function, CBC; watch for neurotoxicity (encephalopathy, myoclonus, seizures) at high levels in renal impairment.
ED Pearl
Strong gram-negative/Pseudomonas coverage but weak against gram-positives — don't use as monotherapy when staph or strep are plausible pathogens.
Symptomatic bradycardia, organophosphate/carbamate poisoning secretions, and selected cholinergic toxidromes.
What’s your dose? — reveal dosing & cautions
ED Dose
Bradycardia: 1 mg IV q3–5 min, max 3 mg. Organophosphate poisoning: 2–5 mg IV, double every 3–5 min until bronchorrhea/bronchospasm improve, then infusion based on effective dose.
Renal Adjustment
No renal adjustment for emergency use.
Contraindications
No absolute contraindication in unstable bradycardia or cholinergic poisoning. Caution in narrow-angle glaucoma/urinary retention for non-emergent use.
Interactions
Other anticholinergics increase delirium/hyperthermia/urinary retention; potassium chloride tablets GI injury risk may increase with slowed motility.
Monitoring
HR, BP, mental status, temperature, secretions/airway in organophosphate poisoning.
ED Pearl
In organophosphate poisoning, the endpoint is drying the lungs, not normalizing the pupils or heart rate — atropine until ventilation improves.
For educational use only. Verify dosing against the FDA label and your institution’s pharmacy resources before administering.
ECG of the Day
Misc
Hyperthyroidism
Unexplained sinus tachycardia or new atrial fibrillation with high LV voltage but no strain should put thyrotoxicosis on your differential.
The Tracing
A thin, tremulous 34-year-old woman presents with palpitations, heat intolerance, and weight loss over two months. She is anxious and diaphoretic. Her heart rate is 132. The 12-lead shows an irregularly irregular rhythm with no discernible P waves and a rapid ventricular response. The QRS voltages are tall enough to meet criteria for left ventricular hypertrophy, yet there is no accompanying ST depression or T-wave inversion of a strain pattern. Scattered through the tracing are nonspecific ST and T-wave changes and an occasional early wide beat. You note her staring gaze and the fine tremor of her outstretched hands as you order labs.
Sinus tachycardia (around 50% of thyrotoxic patients have a resting rate >100 bpm)
Atrial fibrillation with rapid ventricular response (seen in up to 20% of patients)
High left-ventricular voltage meeting LVH voltage criteria without an LV strain pattern
Other supraventricular arrhythmias — premature atrial beats, PSVT, multifocal atrial tachycardia, atrial flutter — plus nonspecific ST/T changes and ventricular extrasystoles
In thyroid storm, atrial tachycardias at rates >200 bpm
Pearls
Atrial tissue is especially sensitive to thyroid hormone, which is why atrial tachydysrhythmias dominate the picture — the ECG changes are driven by sympathetic activation plus direct myocardial stimulation.
High voltage without strain is a useful discriminator: it suggests a hyperdynamic, thyroid-driven state rather than the pressure-overload LVH you'd expect to come with a strain pattern.
Unexplained sinus tachycardia or new AF is itself a prompt to check TSH and T4 — the ECG can be the first clue that sends you to the thyroid.
Pitfalls
Attributing new AF or sinus tachycardia purely to anxiety, pain, or volume status misses occult thyrotoxicosis — the source explicitly recommends thyroid function testing in these patients.
Rates >200 bpm signal possible thyroid storm, a life threat that shouldn't be managed as ordinary rate control alone.
Reading the high voltage as fixed structural LVH can anchor you away from the reversible thyroid driver, especially when the expected strain pattern is absent.
At the Bedside
Send TSH and T4 in any patient with unexplained sinus tachycardia or new AF, and control thyrotoxic atrial tachydysrhythmias with intravenous beta-blockade (titrated IV propranolol boluses or an esmolol infusion) while treating the underlying thyrotoxicosis; escalate to thyroid-storm management when rates exceed 200 bpm.
For educational use only. Verify ECG interpretation against the LITFL entry and your institution’s practice before clinical decision-making.
Case of the Day
From the lead · Transgender Care in the ED, Part 1
Self-Examination
Test Your Understanding
A 25-year-old transgender man taking testosterone presents with pelvic pain and has not menstruated for 10 months. He has a uterus and ovaries and reports sex with a partner who produces sperm. Which statement is most accurate?
BPregnancy testing is indicated if pregnancy is biologically possible.
CTestosterone is a reliable contraceptive when taken daily.
DA pelvic examination is required before pregnancy testing.
Reveal answer
Correct answer · B
Testosterone and amenorrhea do not reliably prevent or exclude pregnancy. Testing should be based on pregnancy potential and exposure; an examination is not a prerequisite.
Study Pace4 topics today; Issue 73 of 94 — Renal and Urogenital / OB/GYN (Weeks 40-41 A)Deadline · June 1, 2026