An Emergency Medicine Broadsheet
·Phoenix·
Est. MMXXVI
Blue Fish Med · Today's Topic
Jack and the Ear Bean
A seemingly minor ear foreign body can become an external-canal laceration, tympanic-membrane perforation, or retained organic material with delayed infection. The first attempt is often the best attempt—if visualization and equipment are adequate.
An 8-year-old boy sits rigidly on the stretcher, one hand clamped over his right ear and the other twisting a baseball cap damp with sweat. His mother says he pushed something into the canal while playing with dried beans, and now he says the room sounds “underwater.” The otoscope shows a pale, smooth object deep in the canal, but the child jerks away before the view is complete. The next attempt has not yet been made.
— What’s your move? Read on.
Before you read
When should you stop bedside attempts and call ENT?
How do you protect the tympanic membrane during extraction?
When to Think of It
Suspect an aural foreign body when a child has unilateral pain, hearing loss, otorrhea, bleeding, or unexplained agitation. Beans, peas, and other organic objects may absorb moisture and expand; button batteries cause rapid liquefactive injury and require immediate removal.
Sick or Not Sick
The key call is safe, complete visualization with a cooperative patient—or not. Deep objects, sharp objects, button batteries, suspected tympanic-membrane perforation, significant bleeding, or an uncooperative child should trigger early ENT involvement and possible procedural sedation.
The First Fifteen Minutes
Stabilize the child and avoid repeated probing; inspect both ears and document the object’s location.
If pain limits examination, give acetaminophen 15 mg/kg PO, maximum 1,000 mg, because analgesia improves cooperation without impairing protective reflexes.
For significant pain or a planned extraction, give fentanyl 1 mcg/kg IV or intranasally, maximum initial adult dose 100 mcg, because rapid analgesia reduces movement; monitor ventilation.
If the canal is clearly intact and the object is nonexpanding, nonbattery, and nonorganic, use the least traumatic visualized technique—instrument extraction, suction, or gentle irrigation.
Do not irrigate a bean, other hygroscopic organic material, button battery, or object with possible tympanic-membrane perforation; water can expand the object or worsen chemical injury.
If procedural sedation is necessary, use ketamine 1 mg/kg IV (repeat 0.5 mg/kg after 5–10 minutes if needed) or 4–5 mg/kg IM when IV access is not practical; it preserves spontaneous ventilation and usually provides immobility. Dosing varies by protocol—check institutional guidance.
If canal trauma produces active bleeding, apply gentle direct pressure with a small cotton pledget; do not blindly pack against an uncertain tympanic membrane.
Definitive Care & Disposition
Remove the object under direct visualization with alligator forceps, curette, suction, or a balloon catheter when appropriate. Stop after one or two unsuccessful attempts, significant canal trauma, worsening pain, or loss of visualization. ENT should remove deep, sharp, battery, or impacted objects and evaluate tympanic-membrane injury. Discharge is reasonable after complete atraumatic removal with a normal membrane and reliable follow-up; arrange ENT follow-up for perforation, retained fragments, canal laceration, infection, or failed removal. Antibiotic ear drops are not routine after uncomplicated extraction; use topical therapy only when directed for contaminated canal trauma or otitis externa.
How This One Kills
Repeated blind attempts drive the object medially, convert a simple extraction into tympanic-membrane perforation, and may leave an expanded bean wedged against the membrane.
The Differential — What Else Looks Like This
Otitis externa — tragal/pinna tenderness without a discrete object; confusing it with a foreign body delays antimicrobial ear care.
Acute otitis media — bulging, opaque tympanic membrane rather than a canal object; instrumentation can injure an already inflamed membrane.
Tympanic-membrane perforation — otorrhea, conductive hearing loss, and an absent membrane segment; irrigation can introduce infection or deeper injury.
Button-battery injury — metallic disc with surrounding gray/black tissue or alkaline drainage; minutes matter because tissue necrosis progresses rapidly.
The Second-Day Story
Toddlers may present only with irritability, sleep disruption, unilateral foul drainage, or reduced response to sound. In an older patient, chronic unilateral otorrhea may be mistaken for otitis externa. A careful bilateral otoscopic examination, including the canal behind the pinna and a search for retained fragments, is more reliable than the history.
Back to Our Patient
Back to the 8-year-old boy with the damp baseball cap: he has a visible organic ear foreign body, but the object is deep and he cannot remain still, so the risk-stratifying decision is that bedside manipulation without sedation is unsafe. After monitoring and analgesia with acetaminophen, he receives ketamine procedural sedation, allowing atraumatic removal under direct visualization without irrigation; the tympanic membrane is intact and hearing improves immediately. He is discharged with return precautions and ENT follow-up only if pain, drainage, fever, or hearing loss develops.
Patient Presentation to Attending
How you’d present this patient on the floor — tight, pertinent positives and negatives, no rambling
“This is an 8-year-old boy with acute right ear pain and decreased hearing after inserting a dried bean into the canal shortly before arrival. He has no fever, vertigo, head trauma, or purulent drainage, but he is unable to tolerate otoscopy or remain still for removal. The visible object is pale and smooth, located deep in the external canal, with no obvious active bleeding, although the tympanic membrane is not yet visualized. I’m concerned about an expanding organic foreign body with potential canal or membrane injury. I’ll stop blind attempts, provide analgesia, obtain appropriate equipment and monitoring, and proceed with controlled removal under procedural sedation with ENT backup.”
Study Directive
Practice identifying which ear foreign bodies are safe versus unsafe for irrigation.
Review and verbally rehearse a pediatric procedural-sedation plan, including monitoring and rescue equipment.
Draw the external auditory canal and mark where medial extraction becomes high risk.
Perform a timed oral presentation for a child with a deep ear foreign body.
Curry SD, Maxwell AK · Otolaryngol Clin North Am, 2023 · PMID 37516654 · cited 3×
Provides a focused guide to safe ear-canal foreign-body extraction, including technique selection, contraindications to irrigation, and when to stop and consult otolaryngology.
More in Today's Issue
3 additional topics
2 of 4
Magnets up the Nose
Nasal magnets can trap the septum or adjacent turbinates between them, producing pressure necrosis, septal perforation, and tissue loss within hours. A calm...
Also known asnasal foreign body · magnet foreign body · nasal magnets
A 4-year-old girl arrives with blood on her upper lip and a metallic glint visible just inside the right nostril. Her father says she was playing with magnetic building beads and suddenly began crying. She is breathing comfortably, but the left nostril now looks swollen and the child keeps rubbing her nose. The object has not yet been touched.
Before You Read
What makes nasal magnets uniquely dangerous?
When is bedside removal unsafe?
What must be examined after the object is removed?
Why It Matters
Nasal magnets can trap the septum or adjacent turbinates between them, producing pressure necrosis, septal perforation, and tissue loss within hours. A calm child with normal oxygenation can still have a surgical emergency.
When to Think of It
Consider a nasal foreign body with unilateral obstruction, epistaxis, foul discharge, wheezing/snorting, or a witnessed insertion. Bilateral metallic objects, paired magnets, or a magnet plus another ferromagnetic object should immediately raise concern for tissue compression.
Sick or Not Sick
The critical call is single, anterior, visible, graspable object versus paired/impacted magnets with ischemic tissue. Bilateral magnets, posterior objects, significant bleeding, severe pain, septal discoloration, or failed removal require urgent ENT management.
The First Fifteen Minutes
Assess airway and breathing; keep the child calm and upright. Avoid blind probing or repeated attempts.
If pain or anxiety limits cooperation, give acetaminophen 15 mg/kg PO, maximum 1,000 mg, because comfort improves stillness.
If the child has substantial pain and IV access is available, give fentanyl 1 mcg/kg IV or intranasally, titrated with respiratory monitoring, because rapid analgesia facilitates controlled extraction.
For a cooperative patient with an anterior object, apply oxymetazoline 0.05%: 1–2 sprays in the affected nostril (adult formulation; pediatric concentration varies), because vasoconstriction reduces edema and bleeding.
If topical anesthesia is needed, use lidocaine 4% topical spray or pledget sparingly, avoiding excessive dosing; it reduces mucosal sensation and reflex movement.
Remove a single visible magnet with bayonet forceps or suction only if it can be grasped without pushing it posteriorly. Do not use another magnet as a retrieval tool when tissue may be trapped between magnets.
If removal requires sedation, use ketamine 1 mg/kg IV or 4–5 mg/kg IM with full airway monitoring; check the institutional pediatric sedation protocol.
Definitive Care & Disposition
ENT should urgently remove paired or deeply impacted magnets, especially when mucosal blanching, ulceration, necrosis, or septal injury is present. After extraction, inspect both nasal cavities and the septum for perforation and retained fragments. Septal hematoma requires urgent drainage by ENT. Uncomplicated removal with intact mucosa can be discharged with saline, avoidance of nose picking, and return precautions; persistent bleeding, infection, tissue injury, or inability to exclude retained magnets warrants ENT observation or admission.
How This One Kills
Two magnets compressed across the septum are mistaken for one anterior foreign body; delayed removal causes pressure necrosis and a septal perforation that may not be obvious until the magnets are separated.
The Atypical Presentation
A child may deny insertion, and a magnet may be hidden by edema, clot, or crust. Weeks later, unilateral foul drainage or recurrent epistaxis may be the only clues. Examine both nostrils and the septum, and obtain imaging or ENT visualization when the object count or location is uncertain.
Back to Our Patient
Back to the 4-year-old girl with the metallic glint: inspection shows two magnets compressing the anterior septum, making this a high-risk bilateral foreign body rather than a routine extraction. She receives analgesia, topical vasoconstrictor, monitoring, and urgent ENT consultation; because she cannot cooperate and the magnets are impacted, ketamine sedation allows controlled separation and removal. The septum is bruised but intact without hematoma, so she is discharged after observation with ENT follow-up and strict return precautions.
Patient Presentation to Attending
“This is a 4-year-old girl with witnessed insertion of magnetic building beads into the nose, followed by epistaxis and nasal pain. She is breathing comfortably without stridor or aspiration symptoms, but there is a metallic object in the right naris and swelling of the left side of the septum. I’m concerned that paired magnets are compressing the septum and causing early ischemic injury. I’ll avoid blind probing, give analgesia and topical vasoconstriction, and involve ENT urgently for controlled removal under sedation if needed. After removal we need to inspect the septum for hematoma, necrosis, and perforation before disposition.”
Study Directive
Review images of nasal magnets, button batteries, septal hematomas, and rhinoliths.
Practice a “single object versus paired magnets” risk stratification script.
Memorize age-appropriate oxymetazoline concentrations used in your institution.
Perform a simulation of failed bedside removal and escalation to ENT.
Key Medications
Acetaminophen: 15 mg/kg PO, maximum 1,000 mg/dose.
Fentanyl: 1 mcg/kg IV or intranasal, titrated with monitoring.
Oxymetazoline: 0.05%, 1–2 sprays per affected nostril in adults; pediatric concentrations and age limits vary—check product labeling and local protocol.
Ketamine sedation: 1 mg/kg IV or 4–5 mg/kg IM; verify institutional dosing.
Pediatric medication dosing must be weight-based; avoid exceeding topical lidocaine limits.
High-Yield Pearls
Bilateral nasal obstruction after magnet play is a compression injury until proven otherwise.
Normal breathing does not make the injury benign; the threatened structure is the septum.
After extraction, look specifically for septal hematoma—not merely whether the object is gone.
The Mimics
Nasal septal hematoma — boggy bilateral septal swelling after trauma; missing it risks cartilage necrosis.
Impacted clot — soft dark material after epistaxis, without a metallic sheen; forceful extraction can restart bleeding.
Rhinolith — chronic foul unilateral discharge and hard calcified mass; acute magnet injury is more time-sensitive.
Button battery — round metallic object with caustic tissue injury; confusing it with a magnet delays immediate removal.
Board Question
A child has two magnets visible in opposite nasal passages with a pale, compressed septum between them. What is the most appropriate management?
AObserve and recheck in 24 hours
BIrrigate both nares
CUrgent controlled removal with ENT involvement
DApply nasal steroid and discharge
Reveal answer
Correct: C
Paired nasal magnets can cause rapid pressure necrosis and septal perforation. They require urgent, controlled removal, often by ENT and sometimes under sedation.
3 of 4
Post-Tonsillectomy Bleed
Post-tonsillectomy hemorrhage can deteriorate abruptly from a deceptively stable presentation to airway obstruction and hemorrhagic shock. Swallowed blood...
Also known aspost-tonsillectomy hemorrhage · tonsillectomy bleeding
A 17-year-old wakes his mother at 2 a.m. with the metallic taste of blood and a basin half full of dark clots. He is sitting forward, pale and frightened, repeatedly swallowing between short breaths. The tonsillar fossae are difficult to see because blood is pooling in the oropharynx, and his blood pressure has not yet been repeated. The suction catheter is still in its wrapper.
Before You Read
Which action comes before the throat examination?
When does a “small” bleed become an airway emergency?
What treatments can temporarily slow hemorrhage while ENT prepares definitive control?
Why It Matters
Post-tonsillectomy hemorrhage can deteriorate abruptly from a deceptively stable presentation to airway obstruction and hemorrhagic shock. Swallowed blood substantially underestimates blood loss.
When to Think of It
Any bleeding, hematemesis, hemoptysis, frequent swallowing, spitting blood, syncope, pallor, or worsening throat pain after tonsillectomy is a hemorrhage until proven otherwise. Primary bleeding occurs within 24 hours; secondary bleeding commonly occurs around days 5–10 as the eschar separates.
Sick or Not Sick
The single decisive call is can the patient protect and maintain the airway while hemorrhage is controlled? Ongoing bright-red bleeding, clots, repeated swallowing, respiratory distress, agitation, shock, or inability to sit forward indicates a high-risk airway and operative emergency.
The First Fifteen Minutes
Place the patient upright and leaning forward, suction continuously, use cardiac/BP/SpO₂ monitoring, obtain two large-bore IVs, and call ENT and anesthesia immediately.
If hypotensive or showing poor perfusion, give balanced crystalloid 500–1,000 mL IV while blood is obtained; crystalloid restores circulating volume but does not replace oxygen-carrying capacity.
Activate a massive transfusion protocol for shock or uncontrolled hemorrhage; give uncrossmatched O-negative packed RBCs when necessary, with plasma and platelets according to local protocol.
If ongoing oropharyngeal bleeding is present, give tranexamic acid 500 mg nebulized (or 1,000 mg nebulized in many adult protocols); it stabilizes fibrin and may temporize mucosal bleeding. Pediatric dosing varies—check institutional protocol.
If the patient is actively bleeding and cooperative, apply gentle direct pressure with gauze to the tonsillar fossa only when this can be done without provoking vomiting; never perform a blind finger sweep.
If intubation is required for airway protection, prepare for a contaminated airway with suction, video/direct laryngoscopy, and the most experienced operator; give ketamine 1–2 mg/kg IV for induction when hypotension is present because it generally preserves sympathetic tone, with paralysis per airway protocol. Dosing varies—check the local RSI guideline.
Avoid oral intake and avoid routine NSAIDs; establish type and crossmatch, CBC, coagulation studies, fibrinogen, and venous/arterial blood gas as clinically indicated.
Definitive Care & Disposition
All significant post-tonsillectomy bleeds require ENT consultation and generally operating-room evaluation, even if bleeding stops. Resuscitation continues with blood products guided by physiology and laboratory results. Recurrent bleeding, anemia, coagulopathy, shock, airway concern, or active hemorrhage requires ICU-level care and operative control. A minor, clearly resolved bleed may still require prolonged monitored observation and ENT-directed disposition; discharge should be exceptional and specialist-approved.
How This One Kills
The patient appears stable because blood is swallowed, then suddenly aspirates a large clot or collapses from occult blood loss during an attempted examination or induction.
The Atypical Presentation
Children may present only with repeated swallowing, vomiting dark blood, pallor, anxiety, or worsening pain. A normal-appearing mouth between episodes does not exclude a clot hidden in the fossa. Ask the caregiver how many basins, tissues, or episodes of swallowing occurred, and treat any witnessed blood as potentially substantial.
Back to Our Patient
Back to the 17-year-old with the metallic taste and basin of clots: repeated swallowing, active oropharyngeal blood, pallor, and uncertain hemodynamics make this a high-risk post-tonsillectomy hemorrhage. He is placed upright, suctioned, monitored, given IV access and crystalloid while blood is prepared, and receives nebulized tranexamic acid as ENT and anesthesia arrive. He remains able to protect his airway but continues bleeding, so he is taken urgently to the operating room for hemorrhage control and admitted for postoperative monitoring.
Patient Presentation to Attending
“This is a 17-year-old on postoperative day 8 after tonsillectomy with acute oral bleeding, repeated swallowing, and approximately half a basin of dark blood at home. He is pale and anxious, sitting forward, with blood and clot pooling in the oropharynx but no current stridor; his blood pressure is pending repeat and his heart rate is elevated. He has no preceding vomiting or cough to suggest a primary GI or pulmonary source. I’m treating this as an active secondary post-tonsillectomy hemorrhage with potential airway compromise and occult blood loss. I’ve placed him upright on continuous suction and monitoring, established two IVs, sent type and cross, started resuscitation, and called ENT and anesthesia for urgent operative management.”
Study Directive
Rehearse the first 60 seconds of a post-tonsillectomy hemorrhage response aloud.
Review your institution’s tranexamic acid, massive-transfusion, and pediatric RSI protocols.
Practice identifying primary versus secondary hemorrhage by postoperative timing.
Simulate a contaminated airway plan with two suction sources and a backup intubation strategy.
Key Medications
Tranexamic acid: 500 mg nebulized; adult protocols vary between 500–1,000 mg. Verify institutional protocol and pediatric dosing.
Balanced crystalloid: 500–1,000 mL IV bolus, reassessing frequently; use smaller weight-based boluses in children.
Ketamine RSI induction: 1–2 mg/kg IV, with dose adjusted to hemodynamics; verify local airway protocol.
Uncrossmatched RBCs: O-negative when blood is immediately required; activate local massive-transfusion protocol for severe hemorrhage.
Avoid NSAIDs during active post-tonsillectomy bleeding unless specifically directed by ENT.
High-Yield Pearls
Repeated swallowing may be the only visible sign of active bleeding.
A clot in the tonsillar fossa can be a temporary plug; dislodging it without airway and operative backup can precipitate exsanguination.
The patient’s ability to maintain oxygen saturation now does not predict the safety of later airway manipulation.
The Mimics
Hematemesis from a gastrointestinal source — nausea/retching precedes blood and the tonsillar bed is dry; mislabeling it delays GI evaluation.
Hemoptysis — coughing and lower-airway symptoms predominate; treating it as surgical-site bleeding can miss pulmonary hemorrhage.
Postoperative dehydration — tachycardia and poor intake without visible bleeding; assuming dehydration alone delays hemorrhage control.
Peritonsillar abscess — unilateral swelling, muffled voice, and uvular deviation without brisk bleeding; invasive examination can worsen a fragile postoperative site.
Board Question
A 10-year-old presents 7 days after tonsillectomy with repeated swallowing and bright-red blood in the mouth. He is tachycardic but currently maintaining oxygenation. What is the best next step?
ADischarge after a normal-appearing oropharynx
BGive oral ibuprofen and observe at home
CUpright positioning, suction, IV access, ENT/anesthesia consultation, and resuscitation
DBlindly remove the clot with a finger
Reveal answer
Correct: C
Post-tonsillectomy hemorrhage can worsen suddenly, and swallowed blood underestimates loss. Airway preparation, suction, resuscitation, and urgent specialist management take priority over repeated examination or discharge.
Provides a practical framework for deciding which children with post-tonsillectomy bleeding require admission or observation, including those with a reassuring examination after a reported bleed.
Supports nebulized tranexamic acid as a noninvasive temporizing option to control active post-tonsillectomy hemorrhage while securing the airway and arranging urgent otolaryngology care.
4 of 4
Ankle Dislocations
An ankle dislocation threatens skin, neurovascular structures, and the talar blood supply. Prompt reduction can restore perfusion and prevent pressure...
Also known astibiotalar dislocation · ankle dislocation
A 29-year-old warehouse worker lies on the stretcher with his right foot turned almost sideways, the sole facing outward beneath a torn work boot. His toes are cool but pink, and he keeps asking whether he will lose the foot. The deformity is obvious; the dorsalis pedis pulse is faint and difficult to compare with the other side. The reduction kit has not yet been opened.
Before You Read
Which finding makes reduction immediate rather than delayed for imaging?
What must be documented before and after reduction?
When does an ankle dislocation require operative management?
Why It Matters
An ankle dislocation threatens skin, neurovascular structures, and the talar blood supply. Prompt reduction can restore perfusion and prevent pressure necrosis even before the fracture pattern is fully defined.
When to Think of It
Suspect ankle dislocation after high-energy twisting, sports injury, fall, or motor-vehicle trauma when there is gross deformity, inability to bear weight, a displaced talus, skin tenting, or abnormal foot alignment. Most are fracture-dislocations, even if the initial radiograph appears to show little fracture.
Sick or Not Sick
The crucial call is neurovascular compromise or threatened skin. Absent pulses, worsening sensation, severe pain out of proportion, rapidly increasing swelling, open injury, or skin tenting mandates immediate reduction—do not delay for radiographs.
The First Fifteen Minutes
Expose the limb, remove constrictive footwear, assess and document pulses, capillary refill, motor function, sensation, skin integrity, and open wounds.
If the patient is in severe pain, give fentanyl 1 mcg/kg IV, titrated in 25–50 mcg increments in adults, because rapid analgesia facilitates gentle reduction; monitor ventilation.
If reduction is needed urgently and the patient is physiologically stable, use ketamine 1 mg/kg IV, repeating 0.5 mg/kg as needed; it provides dissociative analgesia while generally preserving spontaneous breathing. Check local sedation protocol.
An alternative is propofol 0.5–1 mg/kg IV, titrated in small increments, because it provides rapid sedation; hypotension and apnea are common, so use only with full airway readiness. Dose varies by patient and protocol—check a reference.
Perform longitudinal traction with reversal of the deformity and gentle correction; avoid forceful repeated manipulation.
If an open fracture or gross contamination is present, give cefazolin 2 g IV (3 g if body weight ≥120 kg) as soon as possible, because early gram-positive coverage reduces infection risk; add broader coverage according to wound and institutional protocol.
If there is a contaminated open wound and tetanus status is incomplete or unknown, give Tdap 0.5 mL IM; add tetanus immune globulin 250 units IM for dirty wounds when the primary series is incomplete or unknown.
Definitive Care & Disposition
Obtain post-reduction radiographs, including the ankle and tibia/fibula when indicated, and repeat the neurovascular examination. CT is often needed for posterior malleolar, pilon, talar, or complex fracture patterns. Splint in a well-padded posterior and stirrup splint, elevate, and keep non-weight-bearing. Open injuries, persistent malalignment, irreducible dislocation, unstable fracture-dislocation, neurovascular injury, compartment syndrome, or threatened skin require urgent orthopedic operative management. Even a successfully reduced closed dislocation generally requires orthopedic consultation and admission or expedited surgical follow-up because swelling and instability are substantial.
How This One Kills
Imaging is obtained before reducing a pulseless or skin-tented ankle, allowing prolonged vascular compression and converting a salvageable injury into ischemia, necrosis, or amputation risk.
The Atypical Presentation
Patients with diabetes, neuropathy, intoxication, or vascular disease may report surprisingly little pain and have an unreliable sensory examination. A closed skin surface does not exclude deep vascular injury. Compare pulses and Doppler signals with the uninjured side, document objective findings, and repeat them after every manipulation.
Back to Our Patient
Back to the 29-year-old warehouse worker with the foot turned outward: his faint pulse, cool toes, and threatened alignment make this an immediately reducible ankle fracture-dislocation. He receives monitored analgesia and ketamine sedation, undergoes prompt longitudinal traction and reduction, and regains a strong dorsalis pedis pulse with improved warmth and sensation. Post-reduction films show an unstable fracture-dislocation; his ankle is splinted, he remains non-weight-bearing, and orthopedics admits him for operative planning and swelling management.
Patient Presentation to Attending
“This is a 29-year-old man with a high-energy right ankle injury and gross deformity after a workplace accident. His foot is laterally displaced with skin tenting, cool but pink toes, and a faint dorsalis pedis pulse; motor function is limited by pain, and sensation is reduced over the dorsum compared with the other side. There is no obvious open wound, but the skin is at risk. I’m concerned about a neurovascularly threatened ankle fracture-dislocation, so I recommend immediate reduction under procedural sedation without waiting for radiographs. I’ll repeat and document the neurovascular exam after reduction, obtain post-reduction films, splint him, and involve orthopedics for admission and operative planning.”
Study Directive
Practice the complete pre- and post-reduction neurovascular examination until it takes less than one minute.
Review reduction maneuvers for lateral, posterior, and anterior ankle dislocations.
Memorize your institution’s adult sedation and open-fracture antibiotic protocols.
Work through three radiographs distinguishing ankle, subtalar, and talar dislocations.
Key Medications
Fentanyl: 1 mcg/kg IV, titrated; monitor respiratory status.
Ketamine procedural sedation: 1 mg/kg IV, repeat 0.5 mg/kg as needed; verify institutional protocol.
Propofol: 0.5–1 mg/kg IV, titrated; meaningful variability exists, so check institutional sedation guidance.
Cefazolin: 2 g IV; 3 g IV if ≥120 kg for open fracture prophylaxis.
Tdap: 0.5 mL IM when indicated.
Tetanus immune globulin: 250 units IM for dirty wounds with incomplete or unknown immunization.
Pediatric sedation, antibiotic, and tetanus dosing differs; use weight-based protocols and a dosing reference.
High-Yield Pearls
“Pink” does not equal perfused; compare pulses, Doppler signals, temperature, capillary refill, motor function, and sensation.
A normal pulse after reduction does not make the injury stable; fracture pattern and soft-tissue swelling determine disposition.
Always document neurovascular status before and after sedation, reduction, and splinting.
The Mimics
Ankle fracture without dislocation — deformity is less dramatic and talar alignment is preserved; treating it as a dislocation can cause unnecessary sedation.
Subtalar dislocation — foot is displaced beneath the talus while the ankle mortise may remain aligned; incorrect reduction maneuvers worsen injury.
Tibial shaft fracture with rotational deformity — tenderness and instability extend proximally; missed injury changes imaging and operative planning.
Compartment syndrome — escalating pain with passive stretch and tense compartments; reduction alone does not treat the ischemic process.
Board Question
A patient with an ankle fracture-dislocation has an absent dorsalis pedis pulse and skin tenting. What is the next step?
ACT angiography before any intervention
BImmediate reduction followed by repeat neurovascular examination
CApply ice and obtain outpatient orthopedic follow-up
DPerform an ankle-brachial index before reduction
Reveal answer
Correct: B
A pulseless or skin-threatened ankle requires immediate reduction to restore alignment and perfusion. Imaging and vascular assessment follow reduction if the pulse does not return or vascular injury remains suspected.
Provides an ED-focused framework for urgent neurovascular assessment, prompt reduction, post-reduction imaging and immobilization, and orthopedic disposition of ankle dislocations.
Yesterday’s Differential
The daily puzzle — from editions past
A quick test of recall from prior editions. Commit to an answer before you check.
From yesterday's edition
A 68-year-old man presents for a routine pre-operative evaluation, asymptomatic. Vitals are unremarkable. What’s the diagnosis, and the first move?
Check your answer
Left Axis Deviation. Treat left axis deviation as a trigger to hunt for its cause on the same tracing and the clinical context — look for left anterior fascicular block, LBBB, LVH, inferior MI, ventricular ectopy, a paced rhythm, or WPW — and let the identified driver, not the axis label itself, direct disposition and further workup.
From the September 2 edition
Today, three days ago: Ondansetron. What’s the adult ED dose, and the contraindication you’d most regret missing?
Check your answer
4 mg IV/ODT/PO once; may repeat based on response. Higher or repeated dosing increases QT considerations; use local pregnancy/pediatric protocols when relevant. Known hypersensitivity; congenital long QT or high-risk QT prolongation caution; avoid with apomorphine due hypotension/loss of consciousness risk.
From the August 26 edition
A 6-year-old requires painful fracture reduction. He is alert, hemodynamically stable, and has no significant comorbidities. Which medication best provides dissociative analgesia and immobility while generally preserving spontaneous respiration?
AMidazolam
BKetamine
CFentanyl
DFlumazenil
Reveal answer
Correct · B
Ketamine. Ketamine produces dissociative anesthesia with analgesia and usually preserves spontaneous ventilation and airway reflexes, though monitoring and rescue capability remain mandatory. Midazolam and fentanyl do not reliably provide adequate procedural immobility alone and can depress ventilation.
Journal Watch
From the FOAMed wire
Notable posts and reviews from the last week, ranked by relevance to today’s lead and source trust.
A 28-year-old female presents with fatigue, bruising, and vaginal bleeding. She reports a history of heavy menstrual cycles; however, this cycle has been significantly worse. Over the past week, she has experienced dizziness, generalized fatigue, and increased bruising. She...
Podcast Picks
Two for the shift
Critical Care Perspectives in Emergency Medicine2026-09-01
Brain damage and death from ischemic encephalopathy is common after cardiac arrest. Animal studies and limited observational studies suggest that high oxygen exposure after ROSC may result in neuronal death. As such, it is hypothesized that limiting oxygen exposure (conservative oxygen) many attenuate reperfusion injury and improve outcomes. At present,...
Source
Critical Care Perspectives in Emergency Medicine
Published
2026-09-01
Host
Critical Care Perspectives in Emergency Medicine, Critical Care Perspectives in Emergency Medicine
Dr. Cho Espinosa—surgical intensivist, OB-GYN, and flight doc—joins me to overhaul how we handle catastrophic post-partum hemorrhage. Moving past standard ivory-tower guidelines, Sho dives deep into high-yield...
Annals Journal Watch
From the specialty flagship
One recent paper from Annals of Emergency Medicine worth knowing on shift, chosen for practice impact — independent of today’s topics.
Cozzi G, Zago A, Di Mascio AJ, et al. · PMID 42678315 · cited 1×
An observational evaluation of continuous dexmedetomidine for maintaining sedation in severely agitated adolescents addresses a difficult, high-risk ED management problem. It may offer an option when initial medications fail, but requires close cardiorespiratory monitoring and protocolized use.
Bottom line → For refractory adolescent agitation, check whether your ED has a monitored dexmedetomidine pathway rather than improvising an infusion at the bedside.
Pharmacology Corner
Two drugs for the shift
One antimicrobial and one other ED workhorse — selected daily, with sources and last-reviewed dates so every dose is cross-checkable.
Antimicrobial of the Day
Cefdinir
Third-generation oral cephalosporin
Indication
Acute otitis media, acute bacterial sinusitis, community-acquired pneumonia, pharyngitis, and uncomplicated SSTI when an oral cephalosporin is appropriate.
What’s your dose? — reveal dosing & cautions
ED Dose
300 mg PO q12h or 600 mg PO once daily x5–10 days. Pediatric: 14 mg/kg/day (max 600 mg/day) divided q12–24h.
Renal Adjustment
CrCl < 30 mL/min: 300 mg PO once daily. HD: 300 mg every other day.
Contraindications
Cephalosporin hypersensitivity.
Interactions
Iron and aluminum/magnesium antacids reduce absorption — separate by ≥ 2 h. Probenecid increases levels.
Monitoring
GI tolerance; clinical response.
ED Pearl
Co-administration with iron causes harmless brick-red stools from a non-absorbable iron–cefdinir complex — reassure the family rather than working it up as a GI bleed.
Procedural sedation, post-intubation sedation, refractory status epilepticus infusion in ICU context, and induction alternative in selected hemodynamically stable patients.
What’s your dose? — reveal dosing & cautions
ED Dose
Procedural sedation: 0.5–1 mg/kg IV, then 0.25–0.5 mg/kg increments. Post-intubation sedation: commonly 5–50 mcg/kg/min infusion titrated to sedation/BP.
Renal Adjustment
No renal adjustment; context-sensitive accumulation with prolonged infusion.
Contraindications
Hemodynamic instability/shock relative, egg/soy allergy no longer absolute in many policies but check local guidance, hypersensitivity.
Interactions
Opioids/benzodiazepines/other sedatives increase respiratory and hemodynamic depression.
For educational use only. Verify dosing against the FDA label and your institution’s pharmacy resources before administering.
ECG of the Day
Rhythm
Multifocal Atrial Tachycardia (MAT)
An irregularly irregular tachycardia with at least three P-wave shapes in one lead is MAT — a marker of a sick, decompensating patient.
The Tracing
A 74-year-old man with severe COPD is in respiratory distress from an exacerbation, on continuous nebulized beta-agonists and looking exhausted. The monitor shows a fast, irregular rhythm, and you order a 12-lead to sort out whether this is atrial fibrillation. On the tracing the rhythm is irregularly irregular at about 130 bpm — but there clearly are P waves, and they keep changing shape. Studying a single lead you count at least three distinct P-wave morphologies, each with its own PR interval, and the baseline between them is flat with no flutter waves. There is right axis deviation with a dominant R wave in V1. His potassium is low and he is tiring.
Irregularly irregular rhythm with varying P-P, PR and R-R intervals
At least 3 distinct P-wave morphologies in the same lead
Isoelectric baseline between P waves (no flutter waves), with no single dominant atrial pacemaker
Frequently accompanying signs of COPD such as right axis deviation and a dominant R wave in V1 (cor pulmonale)
Pearls
MAT is a transitional rhythm between frequent PACs and atrial flutter/fibrillation — the presence of organized but polymorphic P waves is what separates it from AF.
It is a barometer of severe underlying illness: developing MAT during an acute illness carries roughly 60% in-hospital mortality, with death from the illness, not the arrhythmia.
Because the driver is increased atrial automaticity from hypoxia, hypercarbia, beta-agonists, theophylline, and electrolyte depletion, fixing the patient fixes the rhythm.
Pitfalls
Miscalling MAT as atrial fibrillation and reaching for rate/rhythm control or anticoagulation instead of treating the respiratory failure.
Treating the rate directly and aggressively rather than the cause — the arrhythmia typically resolves once the underlying disorder is corrected.
Overlooking correctable contributors: hypokalemia and hypomagnesemia (often from diuretics and beta-agonists) feed the automaticity.
At the Bedside
Treat the underlying illness — optimize oxygenation and ventilation in the COPD/CHF exacerbation and replete potassium and magnesium — rather than targeting the rhythm; recognize MAT as a red flag for a critically ill patient who needs escalated care.
For educational use only. Verify ECG interpretation against the LITFL entry and your institution’s practice before clinical decision-making.
Case of the Day
From the lead · Jack and the Ear Bean
Self-Examination
Test Your Understanding
A 6-year-old has a dried bean lodged deeply in the external auditory canal. He cannot tolerate extraction attempts. Which is the best next step?
AIrrigate the ear with warm water
BRepeatedly probe the bean until it is dislodged
CProcedural sedation with controlled extraction or ENT removal
DDischarge with topical antibiotic drops
Reveal answer
Correct answer · C
Organic materials can swell with irrigation, and repeated attempts increase canal and tympanic-membrane trauma. Deep, poorly tolerated objects should be removed under controlled conditions, often with sedation or ENT assistance.
Study Pace4 topics today; Issue 53 of 94 — HEENT / Orthopedics (Weeks 29-30 A)Deadline · June 1, 2026