An Emergency Medicine Broadsheet
·Phoenix·
Est. MMXXVI
Blue Fish Med · Today's Topic
Wrist Fracture / Wrist Dislocations
Missed wrist dislocations and perilunate injuries can rapidly cost median nerve function and carpal viability. A “simple fall” can become a hand-salvage emergency if the reduction is delayed or incomplete.
A 68-year-old woman comes in after slipping on wet tile, her left wrist held close to her chest like it might shatter if she breathes wrong. The joint is swollen fast, the skin already shiny, and every pulse of movement sends a sharp wince up her face. Her fingers are pink, but she says two of them feel “weirdly asleep,” and she won’t let anyone get near the hand. The x-ray is not yet in view, and the wrist is sitting there in a shape that does not look negotiated.
— What’s your move? Read on.
Before you read
What deformity or x-ray finding should make you worry about perilunate injury?
Which wrist injuries need urgent reduction in the ED before anything else?
When to Think of It
Any painful swollen wrist after FOOSH, especially with deformity, limited motion, median nerve symptoms, or abnormal carpal alignment on x-ray. Enter the diagnosis immediately when pain seems out of proportion to a “sprain,” the hand is numb, or the wrist looks angulated or “extended and stuck.”
Sick or Not Sick
The one call that matters most: Is the hand neurovascularly intact and is there a true dislocation/perilunate injury requiring urgent reduction? If pulses, cap refill, motor/sensation, and alignment are all okay, you have time for imaging and splinting; if not, reduce now and escalate.
The First Fifteen Minutes
Severe pain before imaging or reduction → fentanyl 1–2 mcg/kg IV or hydromorphone 0.5–1 mg IV for analgesia, because pain control improves exam and allows reduction.
Marked deformity, neurovascular symptoms, or suspected dislocation/perilunate injury → urgent closed reduction under procedural sedation or hematoma block; for hematoma block, lidocaine 1% 5–10 mL without epinephrine into the fracture hematoma, because local anesthesia can facilitate reduction while avoiding full sedation in some patients.
Procedural sedation if needed for reduction → propofol 0.5–1 mg/kg IV bolus, titrated, or ketamine 1–2 mg/kg IV; dose choice depends on hemodynamics and local practice.
After reduction with persistent swelling or concern for instability → repeat analgesia as needed and immobilize in a sugar-tong splint or well-padded volar splint, because it prevents redisplacement and limits forearm rotation.
Open fracture, skin tenting with threatened skin, or gross contamination → cefazolin 2 g IV now (add gentamicin 5–7 mg/kg IV for severe contamination/high-energy open injuries per protocol), because early antibiotics reduce infection risk.
Definitive Care & Disposition
Get post-reduction x-rays. Suspected perilunate dislocation, lunate dislocation, irreducible fracture-dislocation, persistent median nerve deficit, or open fracture needs urgent hand/orthopedic consultation; many require operative fixation. Most reduced, neurovascularly intact, closed distal radius/ulna injuries can go home in a splint with elevation, return precautions, and expedited ortho follow-up in 3–7 days.
How This One Kills
The classic failure is calling a perilunate dislocation a “sprain” or “distal radius fracture,” discharging it splinted but unreduced, and letting median nerve compression or carpal ischemia worsen overnight.
The Differential — What Else Looks Like This
Simple wrist sprain — normal alignment and benign function; confusing it with a fracture-dislocation delays reduction and pain relief.
Scaphoid fracture — snuffbox tenderness with often normal initial x-ray; confusing it with a wrist sprain misses the need for thumb spica and follow-up imaging.
Colles distal radius fracture — dorsal angulation after FOOSH; confusing it with perilunate injury misses the carpal malalignment and urgent reduction requirement.
Carpal tunnel syndrome — isolated median nerve symptoms without traumatic deformity; confusing it with acute wrist dislocation can distract from the structural injury.
The Second-Day Story
Older adults, intoxicated patients, and people with neuropathy may underreport pain and present with only swelling, refusal to use the hand, or subtle numbness rather than an obvious deformity. High-energy trauma can also produce “normal-looking” skin over a severe carpal injury, so the exam must include median nerve testing, cap refill, and a deliberate look at the lateral x-ray for carpal alignment.
Back to Our Patient
Back to our patient: the 68-year-old woman with the shiny, swollen wrist and “sleepy” fingers has a traumatic wrist fracture-dislocation until proven otherwise. On the bedside exam, the priority is to document pulses, cap refill, and median nerve function, then move fast to x-ray and reduction if alignment is off or neurovascular symptoms persist. Her pain is severe enough to justify IV analgesia, and if the wrist is dislocated or perilunate, she needs urgent closed reduction with sedation or a hematoma block, followed by a sugar-tong splint and repeat films. If post-reduction neuro exam is normal and the injury is closed, she can go home with close hand/ortho follow-up; if median nerve deficit or instability remains, she stays for urgent hand surgery.
Patient Presentation to Attending
How you’d present this patient on the floor — tight, pertinent positives and negatives, no rambling
“68-year-old woman with a ground-level fall and severe left wrist pain and deformity. She’s got rapid swelling, limited range of motion, and tingling in two fingers, but her hand is still pink with brisk cap refill and palpable radial pulse. No open wound, no other major injuries, and I’m most concerned about a wrist fracture-dislocation or perilunate injury rather than a simple sprain. I’ve given IV pain control, checked median nerve function, and I’m getting x-rays now with plan for urgent reduction if alignment is abnormal. If post-reduction neurovascular exam is stable, I’ll splint in a sugar-tong and arrange hand/ortho follow-up; if not, I’m calling hand surgery.”
Study Directive
Draw the lateral wrist alignment landmarks from memory: radius, lunate, capitate, third metacarpal.
Practice reading 5 wrist x-rays and labeling distal radius fracture, perilunate dislocation, and lunate dislocation.
Rehearse the post-reduction checklist: pain, median nerve, pulses, cap refill, repeat films, splint, consult.
Review the ED reduction approach and sedation options in your institution’s protocol.
Heineman N, Do DH, Golden A · J Hand Surg Eur Vol, 2023 · PMID 37704022 · cited 5×
Carpal dislocations are easily missed on wrist radiographs, and this review reinforces the ED priorities: scrutinize carpal alignment, assess median nerve function, immobilize/reduce when appropriate, and obtain urgent hand surgery consulta
Osteomyelitis is easy to dismiss as cellulitis or an ulcer, but delays allow bone destruction, bacteremia, and amputation. In diabetics, pressure ulcers,...
A 57-year-old man with diabetes sits barefoot on the stretcher, a draining ulcer on the ball of his foot staining the gauze rust-red. The toe around it is warm and swollen, but the pain is oddly muted, and he shrugs when asked how long it’s been there. He says the wound has been “going on forever,” and now he feels tired and a little feverish, like something has moved deeper than skin. The story is still waiting on the bone.
Before You Read
When does a chronic wound become bone infection until proven otherwise?
Who needs admission, imaging, and IV antibiotics right away?
What bedside clues point to osteomyelitis before the scan?
Why It Matters
Osteomyelitis is easy to dismiss as cellulitis or an ulcer, but delays allow bone destruction, bacteremia, and amputation. In diabetics, pressure ulcers, puncture wounds, and hardware infections are common traps.
When to Think of It
Think osteomyelitis in chronic nonhealing wounds over bone, diabetic foot ulcers, puncture wounds through shoes, pressure injuries, post-op or hardware sites, persistent focal bony pain, and draining sinus tracts. Fever may be absent; elevated ESR/CRP, focal tenderness, and a positive probe-to-bone test should raise suspicion.
Sick or Not Sick
The one call that matters most: Is the patient septic, systemically ill, or does the limb have deep space infection/necrosis needing admission and urgent source control? Stable patients can often be worked up with imaging and cultures; unstable patients need resuscitation, IV antibiotics, and surgical consultation now.
The First Fifteen Minutes
Sepsis, hypotension, or toxic appearance → 30 mL/kg crystalloid bolus and broad-spectrum IV antibiotics immediately after blood cultures, because early perfusion support and antimicrobials improve outcomes.
Concern for severe diabetic foot infection, polymicrobial infection, or necrotizing process → vancomycin 15–20 mg/kg IV plus piperacillin-tazobactam 4.5 g IV; add clindamycin 900 mg IV if necrotizing toxin-mediated infection is possible, because coverage must include MRSA, gram-negatives, and anaerobes.
Ceftriaxone 2 g IV daily is a common narrower option for selected stable cases with likely hematogenous or gram-negative coverage needs; tailor to source and local susceptibility.
Pain → acetaminophen 650–1,000 mg PO/IV or opioid titration as needed; do not let analgesia delay cultures, imaging, or consults.
If diabetic foot ulcer is present → offload the foot and avoid probing aggressively in the ED, because further tissue trauma worsens spread.
Definitive Care & Disposition
Obtain plain films first if looking for cortical destruction, foreign body, gas, or hardware issues; MRI is the most sensitive test when diagnosis remains uncertain. Blood cultures are indicated if febrile or ill, and bone biopsy/culture is the diagnostic gold standard when feasible. Most confirmed or strongly suspected cases with systemic illness, deep ulcer, hardware involvement, or inability to ambulate require admission, ID/orthopedic/podiatry consultation, and a prolonged antibiotic course; selected stable patients may be discharged only with reliable follow-up and a clear treatment plan.
How This One Kills
The deadly mistake is treating a diabetic foot ulcer as simple cellulitis, giving a short oral course, and missing the underlying bone infection until sepsis or amputation follows.
The Atypical Presentation
Older adults, diabetics, and immunocompromised patients often lack fever and may present with vague malaise, mild drainage, or “just a sore that won’t heal.” Pain can be surprisingly absent in neuropathic feet, so the physical exam and wound history matter more than the temperature curve; if a chronic ulcer probes to bone, osteomyelitis is on the table even with a normal x-ray.
Back to Our Patient
Back to our patient: the 57-year-old man with diabetes and a draining plantar ulcer has classic concern for diabetic foot osteomyelitis. He gets a focused exam for depth, probe-to-bone, perfusion, and systemic illness, plus x-rays and inflammatory markers while we decide if this is stable enough for advanced imaging or clearly infected enough for admission. Because the wound is chronic and he feels febrile and tired, I’d have a low threshold for blood cultures, IV broad-spectrum antibiotics if he looks systemically ill, and podiatry/ortho consultation for source control and possible bone sampling. If he is unstable, he is admitted; if stable but strongly suspected, he still needs urgent follow-up and a plan that does not stop at cellulitis coverage.
Patient Presentation to Attending
“57-year-old man with diabetes and a chronic draining plantar foot ulcer, now with increased swelling, warmth, and fatigue. He denies major trauma, but the wound has been open for weeks and is very concerning for infection extending to bone; pain is muted, which fits his neuropathy. He’s afebrile right now but looks worn down, and I’m checking vitals, probe-to-bone, perfusion, and whether there’s any deep space involvement or necrotizing change. I’ve ordered x-rays, ESR/CRP, and blood cultures given the concern for osteomyelitis. If he’s systemically ill or the foot looks deep/infected, I’ll start broad IV antibiotics and admit with podiatry/ortho and possibly ID involvement.”
Practice the diabetic foot exam: probe-to-bone, perfusion, neuropathy, deformity, drainage, odor.
Compare x-ray vs MRI findings and write down the situations where each is first-line.
Build one empiric antibiotic regimen for stable, severe, and necrotizing-leaning presentations.
Key Medications
Vancomycin 15–20 mg/kg IV for MRSA coverage; monitor renal function and institutional redosing.
Piperacillin-tazobactam 4.5 g IV for broad gram-negative/anaerobic coverage.
Clindamycin 900 mg IV for toxin suppression in necrotizing concern.
Ceftriaxone 2 g IV daily for selected stable cases; verify organism and local susceptibility.
Cefazolin 2 g IV for MSSA-predominant infection when appropriate.
Metronidazole 500 mg IV/PO every 8 hours if anaerobic coverage is needed with another agent.
High-Yield Pearls
A normal early x-ray does not exclude osteomyelitis; bone changes can lag by days to weeks.
Probe-to-bone in a diabetic foot ulcer is a major red flag, not a minor finding.
If you suspect necrotizing infection, add clindamycin early and do not wait for imaging to consult surgery.
The Mimics
Cellulitis — diffuse superficial erythema without focal bony tenderness or probe-to-bone positivity; confusing it with osteomyelitis leads to undertreatment.
Septic arthritis — exquisitely painful joint with limited passive ROM; confusing it with osteomyelitis delays aspiration and source control.
Charcot neuroarthropathy — swollen warm neuropathic foot with minimal pain and deformity; confusing it with infection can lead to unnecessary antibiotics, but missing infection is worse.
Gout — episodic redness and pain with hyperuricemia history; confusing it with osteomyelitis can delay imaging and cultures.
Board Question
A 62-year-old man with poorly controlled diabetes has a chronic plantar ulcer over the first metatarsal head. The wound probes to bone, he has mild swelling and warmth but no fever, and x-ray is unrevealing. What is the most appropriate next step?
AReassure him and discharge with oral cephalexin for 5 days
BDiagnose Charcot foot and avoid further workup
CObtain MRI and arrange close surgical/ID follow-up, with a low threshold for admission if systemically ill
DPerform immediate bedside incision and drainage of the ulcer
Reveal answer
Correct: C
Obtain MRI and arrange close surgical/ID follow-up, with a low threshold for admission if systemically ill A probe-to-bone ulcer is highly concerning for osteomyelitis even if the x-ray is negative. MRI is the best next imaging test, and definitive care often requires multidisciplinary evaluation and prolonged therapy rather than short-course outpatient cellulitis treatment.
Key ED takeaways for children with suspected acute hematogenous osteomyelitis include obtaining blood cultures and inflammatory markers, using MRI when diagnosis or extent is uncertain, and starting empiric anti-staphylococcal therapy when
3 of 4
Closed Fist Injuries
Closed fist injuries are notorious for deep inoculation of skin flora and oral flora into MCP joints, extensor tendons, and cartilage. The wrong closure can...
The Case
A 23-year-old man comes in two days after a bar fight, his knuckles scraped raw and swollen under the fluorescent light. He keeps saying it is “just a punch,” but the third MCP is puffy, the skin is split, and he can barely make a fist without grimacing. The hand smells faintly metallic and unwashed, and when he finally admits where it happened, the story shifts just enough to matter. The hand looks small; the infection does not.
Before You Read
Which “minor” knuckle laceration is actually a human bite until proven otherwise?
What complication is most likely if you close it like a normal wound?
When does this become a surgical and antibiotic problem?
Why It Matters
Closed fist injuries are notorious for deep inoculation of skin flora and oral flora into MCP joints, extensor tendons, and cartilage. The wrong closure can trap infection and destroy the joint.
When to Think of It
Any laceration over the MCP joint after punching a mouth/teeth, especially the 3rd–5th knuckles, should be treated as a human bite/closed fist injury. Look for pain with motion, swelling, purulence, limited ROM, and delayed presentation.
Sick or Not Sick
The one call that matters most: Is there joint penetration, tendon involvement, or established infection? If yes, this is not a simple laceration; it needs irrigation, antibiotics, and hand surgery evaluation, often admission.
The First Fifteen Minutes
Pain → acetaminophen 1,000 mg PO and/or ibuprofen 400–600 mg PO if safe, because analgesia facilitates exam and irrigation.
If contaminated closed-fist injury or suspected human bite → amoxicillin-clavulanate 875/125 mg PO twice daily for outpatient-appropriate cases, because it covers Eikenella, streptococci, staphylococci, and anaerobes.
If infected, deep, or unable to take PO → ampicillin-sulbactam 3 g IV every 6 hours, because it provides broad bite-wound coverage.
If penicillin-allergic → doxycycline 100 mg PO/IV twice daily plus metronidazole 500 mg PO/IV every 8 hours is a common alternative; local protocols vary, so check references if uncertain.
Tetanus update if not current, because puncture/contaminated wounds are tetanus-prone.
Do not close the wound primarily if it is a bite over the MCP, because primary closure traps bacteria.
Definitive Care & Disposition
Irrigate copiously, obtain x-rays to assess for foreign body, fracture, or tooth fragment, and consider orthopedic/hand consultation if joint capsule or tendon involvement is possible. Delayed presentations, purulence, septic arthritis, tenosynovitis, or inability to ensure follow-up usually require admission and operative washout. Keep the hand elevated in a splint, and arrange close hand follow-up for all but the most trivial superficial injuries.
How This One Kills
The classic error is suturing a “knuckle laceration” closed and sending the patient home, only to have septic MCP arthritis or extensor tendon infection declare itself 24–72 hours later.
The Atypical Presentation
Patients often minimize the mechanism, arriving “late” because the wound looked small and the pain seemed manageable. By the time they present, swelling and stiffness may obscure the original puncture, so the exam must inspect the wound with the hand in flexion and extension to expose deeper injury and foreign material.
Back to Our Patient
Back to our patient: the 23-year-old with swollen scraped knuckles after a bar fight likely has a closed-fist injury from striking teeth. The wound should be explored with the hand positioned to reveal any deeper puncture, x-rays obtained for tooth fragments or fracture, and the joint/tendon involvement assessed carefully because the third MCP swelling raises concern for deeper inoculation. If there is any evidence of bite penetration or infection, he gets antibiotics right away, the wound is not closed, and hand surgery is involved if the joint capsule or extensor mechanism may be violated. If he is stable and superficial only, he still needs irrigation, tetanus review, elevation, and very close follow-up.
Patient Presentation to Attending
“23-year-old man presenting two days after punching someone in the mouth with swelling and laceration over the MCPs, worst at the third knuckle. He initially minimized the mechanism, but this is concerning for a closed-fist human bite injury with possible joint or tendon involvement. The hand is tender and swollen, but I’m checking active extension, joint motion, and whether the wound tracks deep when the finger is flexed. I’m getting hand x-rays for foreign body or fracture, updating tetanus, and I want to start bite coverage now because this is not a simple laceration. If there’s any capsule penetration, purulence, or limited ROM suggesting septic joint or tendon involvement, I’ll call hand surgery and admit.”
Study Directive
Memorize the empiric antibiotic choices for human bite/closed-fist injuries.
Practice documenting extensor tendon function and MCP joint capsule concern from a hand exam.
Review when hand wounds should be left open versus closed.
Sketch the dorsal extensor mechanism over the MCPs and how a clenched fist can deepen the wound.
Key Medications
Amoxicillin-clavulanate 875/125 mg PO twice daily.
Ampicillin-sulbactam 3 g IV every 6 hours.
Doxycycline 100 mg PO/IV twice daily for penicillin allergy alternative.
Metronidazole 500 mg PO/IV every 8 hours if added for anaerobic coverage.
Tetanus vaccine per immunization status; tetanus immune globulin 250 IU IM if indicated for dirty wounds with inadequate immunization.
High-Yield Pearls
The location matters: dorsal MCP laceration after a punch is a human bite until proven otherwise.
Examine the wound with the hand opened and closed; a tiny skin break can hide a deep tendon/joint violation.
The complication you fear most is septic MCP arthritis, which can destroy function quickly.
The Mimics
Simple abrasion — no puncture over the MCP and no mechanism of striking teeth; confusing it with a bite misses deep inoculation.
Boxer’s fracture — deformity and tenderness over the 5th metacarpal neck; confusing it with a bite misses the need to inspect the wound carefully.
Extensor tendon laceration — loss of active extension; confusing it with a superficial bite delays surgical repair.
Cellulitis — diffuse soft tissue infection without a bite mechanism; confusing it with a closed-fist injury can lead to underappreciating joint penetration.
Board Question
A 29-year-old man presents 18 hours after a fight with a laceration over the dorsal aspect of the third MCP joint. He says he punched another person in the mouth. The wound is mildly swollen, and active extension is painful. What is the best next step?
APrimary closure and discharge with oral cephalexin
BCopious irrigation, start amoxicillin-clavulanate, and avoid primary closure
CSplint only; no antibiotics are needed if x-ray is normal
DImmediate discharge without intervention because the wound is small
Reveal answer
Correct: B
Copious irrigation, start amoxicillin-clavulanate, and avoid primary closure This is a classic closed-fist human bite injury and must be treated as contaminated with risk of deep joint/tendon inoculation. Primary closure increases the chance of trapping bacteria, and amoxicillin-clavulanate provides appropriate oral coverage for typical bite flora.
For suspected fight-bite MCP joint violation, this study supports saline load testing as a potential adjunct when exam and imaging do not clearly establish traumatic arthrotomy, reinforcing the need to identify joint penetration early for o
Tendon injuries are easy to miss because the skin wound can be tiny while the functional deficit is major. Missing the diagnosis means permanent loss of...
A 41-year-old chef arrives with a bandaged finger and a tiny clean-looking cut that keeps reopening every time he reaches for his phone. The hand is smeared with blood and flour, and he says the finger “just won’t straighten right,” though the rest of the hand seems okay. He can flex a little, but one joint lags behind like it lost the signal. The knife is gone, the wound is small, and the function is already telling the story.
Before You Read
Which hand motion tells you a tendon is partially or completely cut?
When can a tendon laceration be repaired later, and when does it need urgent consultation now?
What is the biggest exam mistake in hand lacerations?
Why It Matters
Tendon injuries are easy to miss because the skin wound can be tiny while the functional deficit is major. Missing the diagnosis means permanent loss of motion, trigger problems, and avoidable operative delay.
When to Think of It
Any hand laceration with loss of active flexion or extension, abnormal cascade, pain with active motion, or a finger that will not fully extend/flex should trigger tendon injury concern. Examine each digit independently, including at isolated joints, before anesthetizing if possible.
Sick or Not Sick
The one call that matters most: Is there complete loss of active motion, associated nerve/vascular injury, joint penetration, or gross contamination? Those findings push toward urgent hand consultation and often operative repair; isolated partial injuries may be splinted with expedited follow-up.
The First Fifteen Minutes
Pain → local infiltration with lidocaine 1% without epinephrine, 3–10 mL, or digital block as needed, because a careful exam and wound exploration require anesthesia.
Open laceration with concern for tendon involvement → irrigate copiously, place a sterile dressing, and splint in a position of function; do not aggressively probe a questionable tendon in the ED, because that can worsen damage.
If the wound is contaminated or bite-related → amoxicillin-clavulanate 875/125 mg PO twice daily, because contamination raises infection risk around a repairable tendon.
If penicillin-allergic and contamination is a concern → doxycycline 100 mg PO/IV twice daily plus metronidazole 500 mg PO/IV every 8 hours; verify local protocol if unsure.
Tetanus prophylaxis if needed, because hand lacerations are tetanus-prone.
Definitive Care & Disposition
Flexor tendon injuries, extensor injuries with loss of function, suspected pulley injury, and any laceration with nerve or vascular deficit need hand surgery consultation; some extensor tendon lacerations can be repaired outpatient if partial and well-approximated, but timing matters. Document the exact joint position and motion deficit before splinting. Most patients go home only if neurovascularly intact, the wound is clean, and follow-up is reliable.
How This One Kills
The deadly miss is documenting “moves all fingers” without isolating each tendon, then sewing up a laceration over a cut flexor tendon and sending the patient away with a false sense of security.
The Atypical Presentation
Partial tendon lacerations may preserve some motion, so the patient looks “almost normal” but has pain, weakness, or a lag that appears only against resistance. Children, intoxicated patients, and those seen after splinting can hide deficits; the key is isolated testing of FDS, FDP, and extensor function before you decide the tendon is intact.
Back to Our Patient
Back to our patient: the 41-year-old chef with a small knife cut and inability to fully straighten one finger likely has an extensor tendon laceration. I’d isolate the digit exam to confirm whether active extension is impaired at a specific joint, numbness or vascular compromise is present, and whether the wound is contaminated or joint-adjacent. After anesthesia, irrigation, and careful documentation, the finger should be splinted in a position that protects the tendon, and hand surgery should be contacted if there is complete loss of motion, deep injury, or suspected partial tendon cut that may need repair. If the wound is superficial and function truly intact, he can leave with close follow-up and strict return precautions.
Patient Presentation to Attending
“41-year-old chef with a small knife laceration to the finger and now inability to fully extend one joint. The wound looks small, but when I isolated the digit he had extension lag consistent with possible extensor tendon injury, and I’m checking for sensory and vascular deficits as well. There’s no gross contamination or bite mechanism, but I’m irrigating, anesthetizing for a careful exam, and splinting in a protective position. I’m concerned this is not a simple skin cut and may need hand surgery follow-up or urgent repair depending on whether the tendon is partially or completely disrupted. If there’s any associated joint penetration or neurovascular compromise, I’ll escalate immediately.”
Study Directive
Draw the finger flexor/extensor anatomy and label FDS, FDP, and central slip.
Practice a one-minute isolated tendon exam on each digit.
Memorize the red flags that mandate hand surgery: loss of active motion, nerve deficit, vascular compromise, joint penetration.
Review splint positions for flexor vs extensor injuries.
Key Medications
Lidocaine 1% without epinephrine 3–10 mL for local anesthesia or digital block.
Amoxicillin-clavulanate 875/125 mg PO twice daily if contaminated/bite-related.
Doxycycline 100 mg PO/IV twice daily plus metronidazole 500 mg PO/IV every 8 hours for a common penicillin-allergy alternative.
Tetanus vaccine/TIG as indicated by immunization status and wound risk.
High-Yield Pearls
Test tendons against resistance, not just for “some movement.”
A tiny dorsal laceration can equal a major extensor tendon injury.
Document exact joint-specific deficits before anesthesia if possible; that note often determines follow-up and repair.
The Mimics
Pain inhibition from swelling — function improves with analgesia; true tendon cut persists despite effort, and confusing them leads to missed repair.
Nerve injury — sensory loss may dominate without clear motion loss; confusing it with tendon injury misses the need for sensory mapping and possible exploration.
Joint dislocation — abnormal posture and blocked motion can mimic tendon rupture; confusing it with tendon injury can delay reduction.
Trigger finger — clicking/locking with intact tendon continuity; confusing it with a laceration overstates the need for surgery.
Board Question
A 38-year-old woman cuts the dorsal aspect of her index finger with a kitchen knife. The laceration is small, but she cannot actively extend the DIP joint, though passive extension is intact. What is the most appropriate next step?
AReassure her because the skin wound is small
BTreat as a possible tendon laceration, splint, irrigate, and arrange hand surgery evaluation
CClose the wound primarily without splinting
DDiagnose a sprain and prescribe NSAIDs only
Reveal answer
Correct: B
Treat as a possible tendon laceration, splint, irrigate, and arrange hand surgery evaluation Loss of active extension with preserved passive motion is classic for tendon disruption. Small skin wounds can hide major functional injury, so the ED should protect the digit, avoid missing associated damage, and arrange definitive hand evaluation.
For partial extensor tendon lacerations, management hinges on zone, percent tendon width involved, and extensor lag, helping ED clinicians decide who may be splinted with close hand follow-up versus needing urgent repair.
Real-world outpatient extensor tendon repair outcomes inform ED disposition by supporting referral pathways for selected stable open extensor tendon injuries rather than automatic admission or immediate operative repair.
Yesterday’s Differential
The daily puzzle — from editions past
A quick test of recall from prior editions. Commit to an answer before you check.
From yesterday's edition
A 61-year-old man presents with palpitations. The ECG shows a narrow-complex rhythm at 150 bpm that appears regular. What’s the diagnosis, and the first move?
Check your answer
Atrial Flutter. Assess stability. Cardiovert if unstable. If stable, pursue rate or rhythm strategy per local protocol, evaluate duration and anticoagulation needs, and treat triggers.
From the June 25 edition
Today, three days ago: Tranexamic Acid (TXA). What’s the adult ED dose, and the contraindication you’d most regret missing?
Check your answer
Trauma: 1 g IV over 10 min, then 1 g IV over 8 h (CRASH-2 protocol). Postpartum hemorrhage: 1 g IV over 10 min within 3 h, may repeat once. Topical/nebulized for epistaxis or hemoptysis. Active intravascular thrombosis or thromboembolic disease; subarachnoid hemorrhage (relative — risk vs benefit); known hypersensitivity.
From the June 18 edition
A 62-year-old man with poorly controlled diabetes presents with fever, unilateral facial pain, nasal congestion, and a black eschar on the hard palate. He is hypotensive and confused. Which is the best next step?
AIntranasal oxymetazoline and outpatient ENT follow-up
BIV ceftriaxone and observe for clinical improvement
CIV liposomal amphotericin B and urgent surgical consultation
DOral fluconazole and discharge
Reveal answer
Correct · C
This is classic rhino-orbital-cerebral mucormycosis until proven otherwise. The key is immediate amphotericin plus urgent debridement; delay for imaging or cultures increases mortality.
Journal Watch
From the FOAMed wire
Notable posts and reviews from the last week, ranked by relevance to today’s lead and source trust.
Chris Nickson Flexible Nasopharyngolaryngoscopy Flexible nasoendoscopy (FNE) uses in critical care, indications and contra-indications, technique, anatomy and key findings, and useful videos for learning.
A 34-year-old male presents to the ED about 30 minutes after he slipped on ice while shoveling snow at his house. He currently has a superficial abrasion approximately 6 X 6 cm to his posterior scalp without any laceration present that could be closed with sutures. The...
In this episode, Sam Ashoo, MD and Dr. T.R. Eckler, MD discuss the May 2026 Emergency Medicine Practice article, Emergency Department Evaluation and Management of Serious and High-Risk Infections in the Febrile Returning Traveler . 0:16 — Podcast Introduction 1:08 — Episode Introduction 7:30 — Malaria 13:55 — Dengue 17:33 — Enteric Fever...
Hip complaints are bread-and-butter emergency medicine—but every so often they are anything but straightforward. The obvious shortened, externally rotated leg after a fall is one thing; the patient with acute hip...
Pharmacology Corner
Two drugs for the shift
One antimicrobial and one other ED workhorse — selected daily, with sources and last-reviewed dates so every dose is cross-checkable.
Antimicrobial of the Day
Cefazolin
First-generation cephalosporin
Indication
MSSA and streptococcal skin/soft tissue infection, open fracture prophylaxis per trauma/orthopedic protocols, peri-procedural prophylaxis, and selected cellulitis cases without MRSA risk.
What’s your dose? — reveal dosing & cautions
ED Dose
2 g IV once/q8h; 3 g IV for patients ≥120 kg in many prophylaxis protocols. Open fracture prophylaxis commonly begins with 2 g IV as early as possible.
Renal Adjustment
Adjust interval after initial ED dose when CrCl < 50 mL/min; hemodialysis dosing depends on timing and local protocol.
Contraindications
Severe cephalosporin allergy; caution with immediate anaphylaxis history to beta-lactams.
Interactions
Warfarin effect may increase with prolonged therapy; probenecid increases beta-lactam levels.
Monitoring
Allergic reaction, renal function for repeat dosing, clinical response of cellulitis/wound infection.
ED Pearl
For clean open fractures, early cefazolin is more important than exotic coverage — give it quickly, document time, then tailor based on contamination pattern.
Reversal of opioid-induced respiratory depression; empiric use in undifferentiated coma with pinpoint pupils or hypoventilation.
What’s your dose? — reveal dosing & cautions
ED Dose
0.04–0.4 mg IV titrated to respiratory rate (start low to avoid precipitated withdrawal). 2 mg IN or IM (community dosing). Infusion: start at 2/3 of the effective bolus dose per hour.
Renal Adjustment
No adjustment for renal impairment.
Contraindications
Known hypersensitivity (rare).
Interactions
Can precipitate severe withdrawal and pulmonary edema in opioid-dependent patients. Reverses agonist effect of buprenorphine partially and unpredictably.
Monitoring
Continuous pulse oximetry and respiratory rate for at least 2 h after the last bolus, longer for long-acting opioid ingestions (methadone, buprenorphine, extended-release oxycodone).
ED Pearl
Aim for adequate ventilation, not full alertness — full reversal precipitates withdrawal and can mean a combative, vomiting patient with reaspiration risk. Start at 0.04–0.1 mg IV in dependent patients and titrate up.
For educational use only. Verify dosing against the FDA label and your institution’s pharmacy resources before administering.
ECG of the Day
Conduction
AV Block — Mobitz II
Intermittent dropped QRS complexes without progressive PR prolongation imply infranodal disease and a high risk of complete heart block.
The Tracing
A 76-year-old patient with dizziness has an ECG showing regular sinus P waves at 75 bpm. Most P waves conduct with a constant PR interval, but every third P wave is suddenly not followed by a QRS complex. The conducted QRS complexes are wide with a right bundle branch block morphology. There is no progressive PR lengthening before the dropped beat.
Constant PR intervals in conducted beats before and after a non-conducted P wave
Intermittent dropped QRS complexes without preceding PR prolongation
Often associated with a wide QRS because the block is commonly infranodal
May appear as 2:1 AV block where Mobitz I vs II cannot be proven from PR behavior alone
Can progress abruptly to complete heart block
Pearls
Mobitz II is a pacemaker rhythm, even if the patient looks okay in the moment.
A wide QRS with dropped beats should raise suspicion for infranodal disease.
When the rhythm is 2:1 AV block, you often cannot definitively classify Mobitz I vs II; risk-stratify based on QRS width, symptoms, and clinical context.
Pitfalls
Do not reassure yourself because the ventricular rate is currently acceptable; progression can be sudden.
Do not confuse blocked PACs with Mobitz II. Look for premature, abnormal P waves deforming the preceding T wave.
Atropine may not work well for infranodal block and can occasionally worsen conduction patterns.
At the Bedside
Place pacing pads, monitor continuously, evaluate reversible causes, and admit with cardiology involvement. If unstable, pace and support perfusion rather than waiting for progression.
For educational use only. Verify ECG interpretation against the LITFL entry and your institution’s practice before clinical decision-making.
Case of the Day
From the lead · Wrist Fracture / Wrist Dislocations
Self-Examination
Test Your Understanding
A 34-year-old man falls onto an outstretched hand and presents with wrist pain, swelling, and numbness in the index and middle fingers. Exam shows a swollen wrist with limited motion; pulses are intact. X-ray shows abnormal alignment of the carpal bones with the lunate rotated volarly. What is the next best step?
AApply a volar splint and discharge with NSAIDs
BImmediate closed reduction of the wrist injury
CThumb spica splint and outpatient MRI
DEmergent carpal tunnel release without imaging
Reveal answer
Correct answer · B
Immediate closed reduction of the wrist injury This is a perilunate/lunate dislocation pattern with median nerve symptoms and requires urgent reduction to protect nerve function and carpal perfusion. Splinting alone delays definitive care, and MRI or isolated carpal tunnel release does not address the structural malalignment.
Study Pace* 4 topics today; 64 remaining; Day 27 of 43Deadline · June 1, 2026